Eggs worm in feces

Normally, the worm's eggs are not detected in the stool. In the presence of helminth eggs according to their morphological features, the presence of invasion and the appearance of helminths can be established. In a single routine study, the incidence of helminth eggs in stools in patients with helminthic infestations is relatively low. Therefore, the negative result of a single study of feces on eggs of helminths does not yet indicate a real absence of the disease. Moreover, even negative results of repeated studies of feces on helminth eggs should not be considered a reliable sign of the absence of helminth invasion.

Our body is the ideal habitat for millions of small organisms. Some are so small that their travel from the knee to the navel can be compared to a man's flight to the moon. They splash in the lakes of our saliva, dine in the restaurants of the oral cavity, luxuriate in the nooks of the intestines and travel along the rivers of blood, lymph and urine. But the most unpleasant thing is that many parasites quietly eat us from the inside. ..

Whitworm is a worm, the front part of the body is much thinner and longer( in the form of hair) than the back one. It reminds me of a surgical curved needle with a thread of silk or catgut for sewing surgical wounds. The size( length) of the caudal end in the female is 3.5-5.5 cm, in the male it is 4.5 cm. Parasites infiltrate the mucous membrane with a thin hair-like part and attach, and the body hangs freely. Therefore, when colonoscopy( examination of the colon through the anus), they are sometimes mistaken for polyps. And since they feed mainly on the blood of the host, they make sores, from which the blood oozes. Therefore, when examining a colonoscopy or X-ray, doctors also see ulcers of the intestine. Of course, they put diagnoses: polyposis of the intestine;ulcerative colitis;cancer. Most often, vaginal heads parasitize the cecum( hence appendicitis), with intense invasion can live in other parts of the colon, and in rare cases - and in thin.

The variety of parasites is impressive!

The action of helminths on the human body is diverse. They can cause toxic and toxic-allergic phenomena( ascarids, trichinella), have a mechanical effect, injuring the wall of the intestine;cause bleeding leading to anemia( eg, hookworm), as well as to promote the penetration of pathogenic microbes from the contents of the intestine into the blood;close the lumen of both the intestine and the excretory ducts of the liver and pancreas( ascarids), lead to various metabolic and avitaminosis disorders( avitaminosis B12 when infecting with a wide ribbon).

Parasitizing worms belong to one of two subtypes: round( nematodes) and flat( platodes).The latter, in turn, are divided into tapeworms - cestode and flukes - trematode.

In the feces the eggs of the following helminths are most often found.

Microscopic parasitological methods of laboratory diagnostics are direct methods of detection of helminths, their fragments, eggs and larvae of helminths;vegetative and cystic forms of pathogenic protozoa, the detection and identification of which do not require indirect methods of investigation.

To monitor the effectiveness of intestinal helminthiosis treatment, the stool is examined 1 month after its termination. At the first negative result of feces investigation, sampling is carried out 2 more times with an interval of 2-4 days, after which the final result of the laboratory analysis is given out. In strongyloidosis, control of the effectiveness of treatment is carried out only in the study of bile( even if the parasite was detected by co-proskating methods) 1 month after treatment.

1. Ascaris. During the first 5 days of migration through the human body, the larvae of Ascaris feed from their own resources, and then - the BLOOD OF THE HUMAN.This lasts 2.5-3 months. Then - re-infection and again, 2.5-3 months, feeding blood. At the same time, poisons, toxins, the products of the disintegration of migrating larvae, a huge amount of antigens are released. Antibodies that produce immunity can not cope, B-lymphocytes and phagocytes also decrease. As a result of which immunity is sharply reduced, there is a constant intoxication of a person.

2. Pinworms. Violate the mucosa of the small intestine, sometimes to the muscle layer, the lymphoid tissue disappears and, consequently, the nodules of growth of T-lymphocytes, phagocytes. Immunity decreases.

3. Vlasoglav. Lives in the large intestine for 3-5 years, eats blood. Decreases immunity. According to the WHO( 1988), one parasite consumes 0.005 ml of blood per day, and since there is a lot of this parasite, the losses are up to 100 ml of blood daily.

4. Trichinella. Females penetrate into the lymphatic sinuses and vessels of the intestinal wall, where larvae are born, with the flow of lymph and blood, the larvae migrate to the muscles. In muscles, larvae can live for years. Affect lymphoid tissue, lymph nodes, dramatically reduce immunity.

5. Teniosis, pork thistle. Its larvae can spread to all organs. There are a lot of toxins, poisons. There is a constant intoxication of a person.

6. Dwarf tseleen. Lives in the human body to 20 years, affects lymph nodes. It causes a decrease in immunity.

The connection of helminthic diseases with carcinogenesis requires special consideration. It is established that some trematodes - worm-flukes - can contribute to carcinogenesis. The impact of schistosome Mansoni and schistosome hematobium on the kidney immunological changes associated with the presence of these parasites in the body of patients are factors predisposing to the formation of stones in the kidney and bladder. In turn, the formation of kidney stones against the background of schistosomiasises the risk of tumors. There are numerous observations, indicating the relationship of urogenital schistosomiasis and bladder cancer. Based on the totality of the data, the causative agent of the schistosomal schistosomatosis was assigned to the 1 st group of human carcinogens. In the case of schistosomatosis Manson, there are data on the relationship between this disease and the onset of liver cancer.

In the endemic centers of opisthorchiasis in Thailand and Laos, as well as in the clonorchosis foci in China and Vietnam, the incidence of cholangiocarcinoma( gallbladder and bile duct cancer) among the indigenous population is higher than among the same ethnic groups living in non-endemic areas. Experimental studies have shown that proliferative processes caused by opisthorchias often transform into malignant tumors. A detailed study of clinical and epidemiological materials from Thailand in conjunction with the materials of experimental study allowed the International Agency for Research on Cancer to attribute the pathogen of opisthorchiasis to the 1 st group of human carcinogens.

In Russia, many researchers also noted an increased incidence of bile duct cancer in the territory of the hyperendemic foci of our country of opisthorchiasis caused by feline or Siberian fluke. Carcinoma( cancer) of the liver was found in 0.14-0.9% of all autopsies performed in the Tyumen region, where the highest incidence of opisthorchiasis is recorded. According to other authors, the incidence of primary liver cancer in the Tyumen region is 7 times higher than in other regions of the former USSR.

Pathologoanatomical studies( autopsy) in the Tyumen region. According to them, in the south of the Tyumen region, where opisthorchiasis does not exceed 0.5%, the incidence of liver cancer is 4.4 per 100 thousand of the population. In the center of the Tyumen Region( Khanty-Mansi Autonomous Area), where the average population burden of opisthorchiasis is more than 45%, liver cancer is registered at a frequency of 49.8 per 100 thousand population, or more than 10 times more often. In the Tobolsk region( Ob basin), where the incidence of people older than 15 years is 83%, liver cancer in 1940 was detected in 1.3%, in 1958 - in 3.0 and in 1979 - in 4.6%.

In the Khanty-Mansiysk and Tobolsk regions, the levels of primary liver cancer, pancreatic cancer and bile duct cancer are 3.2 and 13 times higher in comparison with similar indices in hypoendemic regions of the same Tyumen region. Pathological studies undertaken by AA Shane revealed a connection between opisthorchiasis and gallbladder cancer. Cholangiocarcinoma in 146 patients with opisthorchiasis with primary liver cancer was detected in 81.5%, and among 125 patients with primary liver cancer without opisthorchiasis - 34.8%.

New scientific facts require a rethinking of the role of viruses, fungi and worms in human pathology. This role is not limited to the traditional concept of pathogenic microorganisms and worms only as causative agents of parasitic diseases. The presence of these parasites not only leads to the development of specific clinical manifestations. Due to allergization and suppression of immunity, parasites create prerequisites for a decrease in the effectiveness of vaccine prophylaxis and a more frequent occurrence of infectious and non-infectious diseases. Moreover, the cure of the latter is often impossible without the simultaneous carrying out of specific anti-passe therapy.

Diagnosis of helminthic invasions is very difficult, sometimes you have to do 5-9 stool tests and do not get positive results. The above enumerated helminthic invasions in human cancer are diagnosed in the WFD in 70% of cases. Now it is clear why anthelmintic drugs are also immunostimulating. Destruction of parasites leads to a rapid improvement in well-being and restoration of immunity.

In conclusion about the helminthic pathology: treatment should be done not one-time, but courses. But the most important thing is not to treat parasites as peaceful cohabitants!

It is necessary to diagnose them in time and promptly treat them.

Taxocarosis is also called visceral syndrome of the wandering larva.

Here are excerpts from scientific journals.

"It is noted that there are two varieties of toxocar pathogens - toxocar canis( worm of the family of dogs) and toxocara katie( family of felines).Up to 50% of children's sandboxes contain invasive eggs toxocar. They persist in the soil for several years. As a result of human infection, their eggs are affected by the liver, lungs and other organs. There are allergies, toxocarcous tumors of internal organs. Toksokaroz looks like a lung, liver, blood disease, etc., which leads to an erroneous diagnosis of "cancer" »

The article describes a clinical example:" Child 2 years. In the blood, the presence of a high level of ferritin - 700( normal - 250), radiological darkening of the rounded shape in the lower lobe of the right lung, which allowed to suspect the cancer process of the right lung. Thanks to the intervention of parasitologists, suspicion of an oncological process was withdrawn, and the child was treated with a diagnosis of "tumor of toxocarcosis" with vermox tablets. As a result, a complete cure, on radiographs - the norm.

In large cities of Russia, the morbidity caused by worm larvae of the variety of ascarids - toxocar. A survey of urban pets showed that 15% of them are infected with toxocaram. Every day in Moscow, about 54 tons of excrement of dogs are formed. In soil and sand samples of squares and parks, children's playgrounds and sandboxes, eggs of worms are often found. Cats are usually of lesser importance, because they instill their feces. In the soil of eggs for a long time retain viability and vagrancy. The widespread distribution of toxocarias among animals is due to the transmission of the pathogen not only through the alimentary route( through the mouth), but also through the placenta, and through breast milk and the transmission of parasites through their own dogs. "

Patients with toxocarias are not infectious( in contrast to ascariasis), sincetheir body development cycle is incomplete( sexually mature forms are not formed).

Symptoms and course of .Clinical manifestations of toxocarias depend on the localization of parasites and their number. In the clinical course, two forms are distinguished: toxocarosis of the internal organs and ocular toxocarosis.

Toxocarosis of the internal organs is often manifested by recurrent fever for several weeks and even months, with the temperature more often sub-febrile to 38 ° C, less often - febrile( above 38 ° C).There is an increase in lymph nodes, often there is total lymphadenopathy. Most patients with visceral toxocarosis have lung damage in the form of bronchitis and bronchopneumonia. In some cases, bronchial asthma may develop. On radiographs of such patients, multiple or single infiltrates are detected, and the lung pattern is increased.

In 80% of patients, the increase in the liver is determined, in 20% - the enlargement of the spleen. In a third of patients, the disease is accompanied by recurrent vascular or pustular rashes on the skin.

In some cases, toxocarosis occurs with the development of myocarditis, pancreatitis.

The defeat of the central nervous system is observed during the migration of toxocar larvae into the brain and is manifested by convulsions such as "small epilepsy", epileptiform seizures, paresis and paralysis. With toxocarosis, behavioral changes are observed-hyperactivity and affects, psychoses, especially in children.

Mechanism of development and spread of the disease. Infection occurs when ingested invasive toxocar eggs. In the upper part of the small intestine larvae emerge from eggs, which through the mucous membrane penetrate the bloodstream, then enter the liver and the right half of the heart. Once in the pulmonary artery, the larvae continue to migrate and move from the capillaries into the pulmonary vein, reaching the left side of the heart and then carried by the arterial blood to organs and tissues.

Circulating the vascular system, they reach the point where the diameter of the vessel does not allow them to move further( larval diameter 0.02 mm).Here they leave the bloodstream, penetrating into the surrounding tissues. Toxocara larvae are deposited in the liver, lung, heart, kidney, pancreas, brain, eyes and other organs and tissues, retaining viability for months and years, staying in the "dormant" state. Some of them can reactivate and continue migration, another part is encapsulated and gradually destroyed inside the capsule. Migrating the human body, the larvae injure tissues, leaving hemorrhages, necrosis, inflammatory changes. For toxocarosis characteristic is the formation of tumors to 8- 10 cm in the liver, lung and pancreas, myocardium, mesenteric lymph nodes, and brain. Their formation occurs due to mechanisms of connective tissue growth around the larvae.

by releasing larvae masking substance capable of protecting the parasite from the aggression of the host antibody using a complex reaction, toxocara in the human body can survive up to 10 years or more.

The mechanism of the appearance of toxocarose in human eyes has not yet been explained. When infecting a small number of human toxocara larvae observed the development of ocular toxocariasis, which can take the development of the granuloma, uveitis, panoftalmitom, chronic endophthalmitis, an abscess in the vitreous, optic neuritis, keratitis, or the presence of migrating larvae in the vitreous. The most common lesion is granulomatous endophthalmitis, resulting in strabismus and narrowing of the visual fields.

Complications. In cases of toxocarosis, cases of development of severe pneumonia are known, leading to fatal outcomes.

Diagnosis and differential diagnosis. Clinical diagnosis of toxocarias is difficult due to a wide range of clinical manifestations, depending on the intensity of the initial infecting dose and the frequency of repeated infections. An epidemiological anamnesis is essential in the diagnosis. Indication of the maintenance in the family of a dog or intimate contact with cats indicates a relatively high risk of infection by toxocarosis.

The presence of allergy to animal hair is also common in toxocarotic invasion. Do not try to detect parasites and their eggs in the stool - adult worms leave the intestine.

The chances of a lifetime parasitological diagnosis of toxocarias are almost zero, since it is impossible to detect migrating larvae in the body. Histological examination of biopsy specimens from organs does not allow revealing larvae of toxocar and establishing a definitive parasitological diagnosis.

Only in case of WFD study, a reliable response can be obtained, as well as localization and the degree of organ damage.

Currently, more and more surgeons and even oncologists are inclined to believe that there are parasites in the organs. Surgeons, understanding that in these cases is pointless to give to the histological examination in remote tumor capsule, the capsule opened themselves, and saw there twisted worms.

Treatment by the method of orthodox medicine. Vermox is 100 mg 2 times a day for 2-4 weeks. Adults sometimes increase the daily dose to 300 mg. Ditrazine citrate is prescribed at the rate of 2-6 mg per 1 kg of body weight per day for 2-4 weeks. Albendazole( 10 mg / kg of patient weight) for 10-20 days.

With ocular toxocarosis, the same treatment regimens are used as in visceral. It is recommended to check individually the sensitivity to one of these drugs.

It is necessary to test medicines, that is, to check the individual sensitivity of a person to one of the above drugs.

In connection with the complex treatment of medication is required 2-3 times less, which reduces toxicity and side effects. Necessarily at the end of treatment with RFT and medications we perform a body cleansing. Due to the fact that migratory larva toxocar can be localized in the skin, organs, only medication can not cope with them. The special role of HRT is growing.

In the case of eye toxocarias, electronic copies of frequencies and medication are prepared in saline solution. This solution is instilled in the eyes 2-3 times a day. The effect comes much faster.

Vegetative-resonance diagnostics allows to reveal all places of larval localization starting from the brain. And not only to reveal, but also to specify precisely the area of ​​defeat in the organ.

Treating ocular toxacarosis is really difficult. The difficulty lies in the fact that parasites die during treatment, which can cause even more clouding and inflammation of the eye-glass body of the eyes.

Possible course of treatment

1. Purify the body and for a week after that it was fed with vegetarian food. As a result, the larvae of toxocar weaken, and the body will cope with intoxication, that is, inflammation and turbidity of the vitreous humor will not follow.

2. After a week of vegetarian food, prescribe vermox by 1/4 tablets 2 times a day for 3 days. Break 4 days, then 1/2 tablet 2 times a day for 3 days. Break 5 days and then 1 tablet 2 times a day for 3 days.

3. Simultaneously with the reception of the vermox, I would recommend enemas 4-6 l fractional.

Against the backdrop of such treatment, haze is possible, hemorrhage in the eye - do not panic. Go immediately to a 1-2-day fasting on the water with enemas. Then - vegetable juices, vegetarian light foods( cucumbers, carrots, boiled beets) until complete enlightenment in the eyes. Do not end the treatment with this. To completely destroy the larvae, it is recommended to dissolve a grain of wormwood every 3 hours( except night) for 3 days, 3 days break. These courses take place 3 times.

Askaridoz is a helminthic invasion that occurs with severe toxic-allergic reactions, eosinophilic infiltrates and hemorrhages in the tissues of various organs.

Characteristic symptoms of the disease are: upper respiratory tract catarrh, salivation, cramping pains in the navel, lining of the tongue, hypotension, disorders of stool and gastric secretion;dizziness, increased fatigue, absent-mindedness, anxious sleep.

Mechanism of the development and spread of the disease. Of mature eggs swallowed by a human, larvae enter the small intestine, enter the intestinal wall, penetrate the blood capillaries, then migrate to the liver and lungs. In addition to the intestines, heart and lungs, ascarinic larvae are found in the brain, eye and other organs. They are intensively feeding on blood serum and red blood cells.

Complications: intestinal obstruction, appendicitis, purulent angiocholitis, subdiaphragmatic abscess, pancreatitis, asphyxia, hemorrhages to the lungs and hemoptysis.

The main indications for treatment of ascaridosis are: violations of the function of the gastrointestinal tract, a decreased appetite, followed by gluttony, neuroses, eosinophilia of blood( an increase in blood cells), atopic bronchial asthma, abdominal pain, etc.

Treatment of orthodox medicine. Vermox is 100 mg 2 times a day for 3 days. When the larva invades the organs, 3 courses are prescribed for 3 days with an interval between courses of 7 days. Children under 10 years - 50 mg per reception. Pregnant is contraindicated.

Folk way: garlic enema( 2 cloves per 2 liters of water).Seeding by milk( or breathing on boiled milk).

VRD and RFT are indispensable for the localization of ascarid larvae to organs, especially in the brain. If the child has dizziness, nausea, headaches, sometimes short-term loss of consciousness - think about ascariasis. And in the intestine they may not be, so the analysis of stool confirm the absence of ascarids can not. If there is no way to diagnose the WFD, you can designate one course of the Vermox and follow the child's condition. With the improvement, conduct 2 more courses and cleanse the body with children's variants XI and XII according to the book "The practice of purification and recovery of the body."

Complex treatment: HRT, induction therapy, herbs, cleansing of the body lead to getting rid of the ascarids and from the abovementioned diseases.

Pathogen - larval forms of anisakid - nematode anisakis. The final hosts of these nematodes are marine aquatic mammals( cetaceans and pinnipeds), fish-eating birds and predatory fish;intermediate hosts - aquatic invertebrates and fish, mainly herring and cod. The level of affection of fish with anisakids is relatively high: in particular, herrings in the Baltic Sea - about 30%, and in the North Sea - 55-100%.

Anisakid larvae are localized in the intestines of fish and migrate to their muscular tissues as soon as the fish dies. A person becomes infected with the use of crude or insufficiently thermally processed fish and aquatic invertebrates( squids, octopuses, shrimps and other crustaceans and mollusks).

Clinical manifestations and mechanism of disease development. Clinical manifestations of anizakiaosis in humans are caused by eosinophilic granuloma( a seal consisting of conglomerates of eosinophils) containing a worm larva of 20-40 mm in the gastrointestinal tract, usually in the stomach and small intestine. Anisakid larvae can promote the development of acute ulcers with perforation and necrosis of the intestinal wall and the formation of tumors in the intestine, which can lead to a narrowing of its lumen.

Over time, seals - granulomas - proliferate, and clinical manifestations become similar to the symptoms of a tumor of the stomach or intestine. There is a bad patency of food, constipation, alternating with diarrhea, irregular vomiting after eating. Even at this stage, clinical methods of research( X-ray, ultrasound) may not yet show the presence of clear tumors.

Due to the fact that this lesion is more often painless, patients can walk for a long time with an unidentified diagnosis. They rarely address physicians, while treating their complaints for symptomatology is interpreted as poisoning. And only if there is almost complete obstruction of food, they begin to conduct a more thorough examination, and then, when revealing multiple seals, they diagnose "CANCER".Usually this happens in cases where patients first turn to oncologists;they open the abdominal cavity and see that the organs are completely covered with nodes - tumors: peritoneum, small intestine, duodenum, stomach, esophagus, the gallbladder and pancreas are captured. It is a terrible conclusion - inoperable cancer, the abdominal incision is sewn up, and the patient dies of starvation because of the obstruction of food. .. Even if oncologists do the connection( anastomosis) of the upper stomach with a healthy part of the intestine, the worms quickly enter into the anastomosis. The lumen of the anastomosis overgrows with worms, which leads to the death of the host organism. Here are such formidable "oncological" worm-mutants!

In recent years, oncology has seen quite a lot of clinical examples of such a course of the tumor process, but no systematic morphological study of these tumors has been carried out. Some miserable information about this disease appeared only recently - only in 1998, and then in works on medical microbiology. In all likelihood, these are mutant parasites that have appeared in connection with the severe pollution by the caloric washings of the rivers, which led to the adaptation of ascarids to life in edible fishes. Those who like to eat raw, half-baked, cold-smoked and lightly salted( that is, not passed the appropriate heat treatment) fish are at great risk to get various "tenants" - parasites.

Since such patients do not handle complaints for a long time, and then, with full overlap of the intestinal lumen, with the symptoms of the "acute abdomen" get on the "first aid" to the operating table.

Treatment by methods of orthodox medicine. Albendazole 400 mg 2 times a day, 15-20 minutes after meals for 3 days. Carry out 2 cycles with intervals between them for 3 weeks. There can be exacerbations, therefore treatment should be carried out under the control of the attending physician.

Enterobiosis is a helminthic invasion caused by pinworms parasitizing in the lower part of the small and large intestine. Catching pinworms in the appendix and female genital organs is possible.

Pinworms have a mechanical, toxic and allergic effect on the body. Contribute to the development of inflammatory processes in the intestine, perianal region( around the anus), female genital organs and urinary tract.

The main symptoms of enterobiasis are: point itching of the anus, nausea, loss of appetite, dry mouth, cramping abdominal pain, diarrhea with mucus, frequent urge to defecate;headache, insomnia, memory loss, absent-mindedness, convulsive seizures in children;dermatitis, abscess, eczema in the anus, perineum and genitalia, proctitis;vulvovaginitis, pyelocystitis, masturbation, bedwetting.

Structure and spread of the disease. The source of infestation is only a patient with enterobiasis. Eggs, laid by female pinworms on the patient's skin, after 4-6 hours mature and become invasive. They fall on the patient's bed and bed linens, household and office items, and are scattered by flies. Infection of a person occurs when ingested mature eggs of pinworms with food and when drifting them into the mouth and nose along with dust.

In patients with enterobiasis, their own infection often occurs due to contamination of the fingers, hand nails, and the combs of the perianal area with itching( Fig. 28).

Treatment by methods of orthodox medicine. With mild forms of enterobiasis, the elimination of infestation can be achieved by carrying out measures that prevent re-infection. To do this, put an enema for adults at night - 4-5, children - 1-3 glasses of water, adding 1/2 teaspoon of soda to each glass. An enema from the lower part of the large intestines is washed by female pinworms, this prevents itching around the anus, combing and staining the body, clothes and bed linen;The patient should sleep in tight-fitting shorts. Bedding and bed linen should be ironed daily with a hot iron. Clean the room with a damp cloth.

With more severe forms, they resort to medical treatment, prescribing orally. When medicinal treatment of this invasion, hygiene is strictly necessary. The most effective for enterobiosis mebendazole( vermox), pyrantel.

Mebendazole( synonyms: vermox, antioxidant, mebutar, etc.) is administered once to adults and adolescents at a dose of 0.1 g( 1 tablet), to children 2-10 years old by 1 / 4-1 / 2 tablets. With repeated invasion, the treatment is repeated with the indicated doses, for multiple invasions, 0.1 g is prescribed for 3 consecutive days. The drug is contraindicated in pregnancy.

Pirantel is administered once for 10 mg / kg body weight. Take the drug inside( without taking a laxative) 1 time a day( after breakfast) in the form of tablets or syrup. Tablets should be thoroughly chewed before swallowing. The analogue of pyrantel is the domestic preparation of embowins.

Prevention. Careful implementation of sanitary and hygienic measures leads to the elimination of enterobiasis. It is necessary to strictly monitor the cleanliness of the body, dwelling, office space, especially children's institutions.

How often we see that someone is chewing on their nails - even in movies, especially foreign ones. Perhaps they show confusion, but in fact, it is an indicator of the presence of pinworms. Eggs pinworms are hammered under the fingernails, apparently, highlighting something annoying, make a person bite off their nails along with the eggs. Pinworms need to evolve, multiply in humans, and such tiny creatures, we, so big and smart, become zombies. We carry out their will: combing, gnawing our nails, children sometimes eat feces.

Many notice how their children bite their nails. There is a desire to tie their hands so that they do not hold them in their mouths. Know: children are forced to make it pinworms. Treat your children and do not forget to treat the whole family, especially pets.

Appendicitis, peritonitis

I did not want to write about the vagaries until I read the clinical example in JI.I. Taranova, where he clearly described the release of the infiltrate - "tumors" from the abdominal cavity after appendectomy - removal of the appendix. The tumor consisted of whipworms.

Belief in surgery, we then hesitated. But there was not enough knowledge to understand why there are postoperative complications and relapses.

Only now it became clear that pain and inflammation, erosion, ulcers are caused by worms. But how they get into the abdominal cavity, the surgeons should answer. Either during surgery from the colon to the peritoneum, either as a result of fistula formation in the large intestine, or through an inflamed appendix, when the integrity of its walls is already broken.

Trichocephalosis - a human helminth disease caused by vaginal head - is characterized by a chronic course with a predominant gastrointestinal function disorder. The disease was described by Morgagni in the 18th century.

Glaucoma is a common parasite, the number of people invaded by it in the world is about 800 million. Glistami mostly infects children aged 5 to 15 years.

Causes of the onset and development of the disease. Pathogen is a round worm, the anterior part of the body is thin, hairy, and the caudal part is thickened( Figure 29).The ratio of the threadlike part to the thickened part of the female is 2: 1, in the male it is 3: 2.Length of females is 30-35 mm, males - 30-45 mm. Adult specimens of helminths parasitize the cecum and adjacent parts of the large intestine. With massive invasion( the number of worms can reach 3 thousand or more), they affect the entire large intestine to the anus. Here females lay up to 3,500 eggs a day, which with faeces fall into the environment. Eggs must be incubated in the soil for 3 weeks or more, after which the larva acquires contagious properties.

There are observations on the invasion of a dog's woolly dog.

Structure and spread of the disease. Trichocephalus belongs to the group of geogelmintoses( geo - land).The source of the invasion is a sick person. Infection occurs as a result of ingestion of mature eggs with contaminated vegetables, fruits, berries, water.

Mechanism of development and ways of spreading the disease. In the pathogenesis of trichocephalosis, the leading role is played by the traumatic action of the pathogen. When the egg is swallowed, the larva is released into the small intestine and implanted into the villi, after a few days it moves to the large intestine, where after 3 months it reaches maturity. Among the adults, females predominate. The life span of the vaginal head in the intestine is about 5 years. Vlasoglavy with their thin head ends damage the mucous membrane, penetrating to the submucosa base, and sometimes the muscle layer. With intensive infestations, bleeding occurs, the formation of erosion and ulcers in the intestine. It is believed that invasive luner-headed people lose 0.005 ml of blood per parasite per day, so that the presence of more than 800 intestinal worms in children leads to anemia. The intoxication effect on the organism of parasite toxins also has a certain value, but is limited mainly to intestinal tissues, contributing to the development of diarrhea( diarrhea).

Clinical manifestations. For the clinical manifestation of helminthic disease, parasitization of a large number of withers is necessary. It is shown that the symptoms of the disease are observed in patients who release more than 5 thousand eggs in 1 g of feces. Patients complain of worsening appetite, nausea, sometimes vomiting, diarrhea, flatulence, increased salivation. Spastic abdominal pains with localization in the right ileal region, epigastrium or other areas are not uncommon. With intensive infestation, hemoccolitis develops, manifested by pain syndrome, spasms, bloody liquid stools. In these cases, especially in children, during straining efforts during defecation, a prolapse of the rectum can occur, on the mucous membrane of which hundreds of attached parasites are visible. There are observations of cases of appendicitis and peritonitis with the detection of withers in the abdominal cavity. Children suffering from trichocephalosis, lag behind their peers in physical and mental development. Trichocephalus is attributed to the role of a factor contributing to the development of intestinal amoebiasis and its severe course. Trichocephalosis is considered a risk factor for appendicitis. In a number of cases, the course of the disease resembles in its clinical course a nonspecific ulcerative colitis.

The prognosis is usually favorable. It can be burdened by the development of anemia and intestinal complications with intense invasion.

Laboratory diagnosis of trichocephalosis is based on the detection of eggs of worms in feces. With endoscopy( colonoscopy), adult worms can be found in the intestine.

Treatment with orthodox medicine. Vermox( contraindicated in pregnancy, breastfeeding and children under 2 years) is prescribed in a dose of 100 mg 2-3 times a day for 3 days after meals.

Albendazole( contraindications are the same) are used at a dose of 400 mg once;with intensive infestation - in the same dose once a day, 3 days in a row.

Medamin - 60 mg in 3 divided doses after a meal for 3 days. Children doses are reduced to 10 mg per 1 kg of body weight per day.

Contraindicated dairy food, coarse fiber and fat. When re-detecting eggs in the stool or on the diagnosis of WFD, repeat the course of treatment.

All children who are lagging behind in studies, carefully, several times to check for trichocephalosis, and even better - to conduct a diagnosis of the WFD.

Ankylostomidosis is caused by ankylostomias, which often enter the human body with unwashed vegetables, fruits or through water, hands. Therefore, ankylostomidosis is sometimes called a vegetarian disease.

The ultimate host is a person whose adult worms parasitize in the upper sections of the gastrointestinal tract, females lay eggs in the upper part of the small intestine. Eggs come out with feces, and their further development occurs in the soil. The ability to introduce larvae, grown in soil, retains up to 1.5 years. Larvae enter the person in two ways: the first - with vegetables, through the mouth, the second - are introduced through the skin. Their sizes are 0.05-0.07 mm, that is, it is a small, thin invisible needle, and even with a hook at the end. Here such needles are fixed on the skin, then "are injected" and through the blood, lymphatic vessels, wander around the small and large circle of the blood stream in search of clean blood. They are called "wandering larvae".But they are also called "clean bloodsuckers", that is, they do not eat anything or drink anything but blood. Vampires are sent for the purest blood - in the lungs and heart. In the first case, bronchitis, pneumonia, pulmonary infiltrates, pleurisy, tracheitis and laryngitis develop with hoarseness and even aphonia. Then, through the airways, larynx and pharynx, they enter the esophagus and intestine. There are pains in the abdomen, like with peptic ulcer, vomiting, diarrhea and general malaise. In addition to the teeth at the end, hookworms have glands that produce special anticoagulants, which cause prolonged bleeding. When gastroscopy is seen erosion, sores in the stomach, pylorosa( gastric pylorus) and duodenum.

After the diagnosis of the WFD, patients should be treated with deworming-treatment from worms, and when gastroscopy is no longer detected erosion and ulceration, the healing passes quickly and without scars. Adult individuals are able to survive in the intestine up to 14 days.

Migrating to a small circle of blood circulation: heart-lungs-heart, - they can linger for up to 200 days.

With a sufficient number of them per day, they can suck up to 1.5 liters of blood, and in response to release the altered toxic liquid. Particularly difficult to tolerate this invasion of children. They have severe anemia with symptoms of heart failure and fainting. With moderate anemia, general weakness is noted, physical and mental fatigue, dyspnea, tachycardia, pale skin and mucous membranes, puffiness of the face, less often - swelling on the legs. In women, violations of the menstrual cycle, early onset of menopause, men - impotence. Patients with ankylostomidosis, especially children, often eat clay, lime, coal, ash, brick, paper, lick metal objects, salt, soap. Children may be delayed in their physical and mental development.

In places where larvae are introduced through the skin, dermatitis phenomena known as "earth scabies", "soil itch" develop. Skin changes occur after lying on the ground, grass, contaminated by feces with larvae, already appeared from eggs ankylostomide.

If infection occurs through the legs, then rashes are more often observed on the feet, in the interdigital folds. Itching, burning, swelling of the tissues, erythema, papules, vesicles. Skin invasiveness symptoms last about 2 weeks.

Clinical cases, when the larvae of ankylostomide are affected by the heart, is a "balm to our medical hearts", since the outcome of our treatment is always positive. Young people( up to 45 years old) with severe heart problems: IHD, arrhythmias, angina pectoris, with valve stenoses( 7 patients), with directions for pacemakers( 2 patients), with endocarditis( 20 patients) - after 7-10 days of treatment inThe center feels only lightness in the heart, no shortness of breath, even with physical exertion. ECG data are improved, blood pressure is normalized. There is no greater joy for the doctor.

Treatment by the method of orthodox medicine is recommended only under the supervision of a doctor.

Pirantel in a dose of 10 mg / kg once for intensive invasion treatment is carried out for 3-5 days, or Vermox is prescribed for 100 mg 2 times a day for 3 days. On the course - 600 mg. In severe cases, albendazole 400 mg once.

All of the above medicines are contraindicated in pregnancy, breastfeeding, children under 2 years. It is very cautious to prescribe these remedies for heart disease, since when larvae are separated, when they leave the ventricular cavity, severe pains, arrhythmias, a sense of death, dips, "fluttering" of the heart, which our patients say, can appear. Patients are frightened, there may be short-term loss of consciousness, an increase in temperature to 38 ° C.There must be a doctor next to the situation.

Additional medications for these reactions are generally not required;you can work on the points of the heart - the method of judok.

It is known about the mortal danger emanating from trichinella, and the need to identify them as soon as possible. They are viviparous, "hatch" their young in lymphatic ways. "No eggs. On the lymphatic vessels and on the blood vessels, the living "cubs" of Trichinella follow in the genetically remembered and their favorite habitats.

Infection with trichinella occurs when eating meat of pigs and wild animals. Many species of animals are susceptible to trichinosis: pigs, wild boars, dogs, wolves, jackals, cats, bears, rats, hedgehogs.

"Trichinella from parasitization in pigs easily passed to parasitization in humans because of the biochemical similarity of the muscles of a pig and a human. But for a pig they are almost harmless, but for a person they can be deadly, as the mutual adaptation of the parasite and the person has not occurred yet. "Before the diagnosis of the WFD, their place of residence in the tissues of organs was not possible in vivo, except to cut out a piece of the organ and send it to a pathomorphological study of parasitologists. And since they never did before, there was an erroneous conclusion: "If the larvae live only in skeletal muscles in pigs, then in humans they too should live only there, and try to think differently!"

But if in the human body theyare born in the lymphocytes, then they can go to the lymphatic tracts not in the muscles, but in completely different tissues and organs. Larvae of Trichinella migrate into the tissue of the mammary glands, lymph nodes, brain, spleen, myocardium of the heart, thyroid gland, muscular wall of the uterus. Only in 2002 there was a good textbook by A. M. Bronstein, AK Tokmalayev "Parasitic diseases of man: protrzozoi and helminthiases", where much is written about trichinella.

"At present, the incidence of trichinosis in Russia has sharply increased. In particular, in 1995 in comparison with 1985-1986 years - in 3,7-4,9 times. Infection of pigs for every thousand carcasses is 8.9 in the public sector and 125.7 in farms.

In the Moscow region in the past 50 years, 52 outbreaks of trichinosis have been reported. Of these, in 1992-1996 there were 32 outbreaks. Currently, the area of ​​trichinellosis covers almost the entire Moscow region. Often the larvae of Trichinella are found in dogs, cats, wolves, badgers, rodents, etc. "

The larvae of Trichinella are rod-shaped, in the places of damage to the muscles and organs they are surrounded by an inflammatory infiltrate. At the second week they are found not only in skeletal muscles, but also in the myocardium, lungs, kidneys, and the brain. According to WHO( 1996), developing immunopathological reactions lead to severe lesions - myocarditis, meningoencephalitis, pneumonia. Inflammatory processes disappear over time, but after 5-6 weeks they can be replaced by dystrophic ones, the consequences of which disappear only after 6-12 months.

By the end of the 1st week after infection, females of Trichinella, immersed in the mucosa of the predominantly small intestine, are found. In severe lesions, ulcerative necrotic lesions of the intestinal mucosa are recorded, which is always interpreted as an oncological or pre-cancer process, and offers surgical removal of the affected bowel.

Most often they spread to muscle groups with abundant blood supply( diaphragmatic, intercostal, tongue, chewing, oculomotor, cervical, upper and lower extremities).In patients with severe disease, 50 to 100 or more Trichinella larvae per gram of muscle are found. By the end of the third week, the larvae acquire a spiral shape, an intense cellular infiltration is observed around them, where a rigid fibrous capsule( cocoon) is then formed. The process of formation of the capsule is disrupted by excessive damage, as well as under the influence of substances with immunosuppressive properties( glycocorticoids, hormones, etc.), as a result of which the larvae of Trichinella become migratory.

With intensive infestation in the myocardium, a focal-diffuse reaction and dystrophic changes develop;the formation of granulomas and the development of inflammation in the small arteries and capillaries of the brain and meninges.

In connection with the polymorphism of symptoms, trichinosis is sometimes taken for abdominal or typhus, epidemic parotitis, opisthorchiasis, fascioliasis, tetanus, influenza, nephritis, rheumatic carditis and pneumonia. And according to my information, take for oncology nodes in the thyroid gland, breast glands, uterus, lymph nodes. Sometimes there is a recurrent trichinosis.

The first symptoms in the form of nausea, vomiting, loose stool, abdominal pain in some patients( up to 12%) appear in the coming days after consuming contaminated meat and can last from several days to 6 weeks.

But these phenomena are often treated as poisoning.

Depending on the nature of the clinical course, the following forms of trichinosis are distinguished: asymptomatic, abortive, mild, moderate and severe.

In asymptomatic form, the only manifestation may be eosinophilia of the blood( an increase in blood cells).The abortive form is characterized by a short-term( 1-2 days) clinical picture.

The leading symptoms of trichinosis are fever, muscle pain, muscle weakness( first century, then face).In our time, because of the terrible ecology and reduced immunity, eosinophilia of the blood may not be.

Patients with trichinosis have maculopa-bullet rashes on the skin, sometimes acquiring a hemorrhagic character. Subconjunctival and subungual hemorrhages are noted.

A constant symptom of the disease is muscular pain, which in severe course can take a generalized character, accompanied by weakness of the muscles and in some cases lead to deforming adhesions and immobilization of the patient. Muscle pains appear first in the limbs, and then in other muscle groups, including chewing, tongue, pharynx, intercostal, oculomotor.

In lung lesions that develop during the first 2 weeks of the disease, cough is observed, sometimes with an admixture of blood in the sputum;signs of bronchitis;on X-ray images, "volatile" infiltrates in the lungs are defined.

In peripheral blood, usually from the 14th day, eosinophilia, leukocytosis, in the blood serum - hypoproteinemia( reduced protein content), decreased levels of potassium, calcium, increased activity of enzymes( creatinine phosphokinase, ALAT, etc.).Eosinophilia reaches maximum values ​​at 2-4 weeks and can persist at 10-15% for 2-3 months or more. There is a certain parallelism between the height of eosinophilia and the severity of clinical signs of trichinosis. In patients with severe course, eosinophilia in the peripheral blood, on the contrary, may be insignificant or even absent.

Trichinellosis of mild course is manifested by a slight increase in temperature( up to 38 ° C), edema of the eyelids, mild pains in the adyschians of the extremities and the humeroscapular region.

The severity of the course of the disease and its outcome is largely determined by organ lesions that develop at the 3rd-4th week of the disease. Myocarditis is the most important among them. With its appearance, there are tachycardia( rapid heartbeat), hypotension, cardiac rhythm disturbance, development of heart failure. The borders of the heart are enlarged, diffuse lesions of the myocardium, sometimes coronary disorders, are revealed on the ECG.Myocarditis in trichinosis is one of the main causes of death occurring in the period from the 4th to the 8th week of the disease. Following myocarditis, a frequent cause of death is lung involvement with possible involvement of the pleura. The appearance of serous effusion in the pleural cavity, congestion in the lungs is also facilitated by the development of cardiovascular insufficiency. Against this background, a secondary bacterial infection can be attached. The defeat of the central nervous system manifests itself as headache, drowsiness, sometimes delirium, and the phenomena of meningism associated with edematous syndrome. In later terms, more serious disorders are registered in the form of polyneuritis, acute anterior poliomyelitis, myasthenia gravis, meningitis, encephalitis, with the development of psychosis, local paresis or paralysis, coma. There is evidence that 10-20% of patients with trichinosis show changes in the central nervous system, while mortality in the absence of adequate treatment can reach 50%.The recovery period for severe trichinosis may last up to 6 months or more.

Let's give an example, described in the book of GA Akopova "Oncology. Experience in cancer treatment. "

Under the influence of an ointment with the content of iron and minerals, a breast tumor of the size of a chicken egg "opened".The mammary gland rejected the gray fibrous tissue consisting of many threads. When the "rod" was removed, the gaze appeared to be a circular cavity. Inside - black-brown viscous blood serum. And a bundle, vertically located in the tissues of the breast, - worm-like formations in an amount of 26 pieces. Inside the cavity, there was already decay. The "cocoon" itself, in which the parasites lived, had dense edges;the walls, similar to the connective tissue scar, reached a thickness of 0.8-1 cm. The consolidation was formed due to dehydration of the tissues, since these parasites are flukes. Gray fibrous tissue in appearance is very similar to excrement( patchwork) of medical leeches. If full leeches placed in a transparent bottle, after a few days, the "threads" of gray will be clearly visible.

Treatment with orthodox medicine. Treating trichinosis is complex, as it involves specific causative and pathogenetic therapy. The causal therapy is aimed at the destruction of intestinal trichinella, the cessation of their production by larvae, the violation of the encapsulation process and the increase in the death of muscle trichinella. In Russia and abroad for these purposes, mainly use mebendazole( vermox), but they do not have a single treatment regimen. In Russia, it is customary to prescribe vermox in a daily dose of 300 mg: 100 mg 3 times a day for 7-14 days;abroad, higher doses of mebendazole are prescribed: the first 3 days - 200-400 mg 3 times a day, then 400-500 mg 3 times a day for the next 10 days. In recent years, for the treatment of trichinosis, albendazole has been used more often in a daily dose of 10 mg per 1 kg of body weight in 3 doses for 7-14 days. The most effective conduct of causal therapy in the incubation period, when possible prevention of clinical manifestations, or in the early days of the disease, when there are still intestinal Trichinella. During the muscular stage of the disease and encapsulation, the effectiveness of the causal therapy is significantly reduced, and its use during this period can exacerbate the disease.

Pathogenetic treatment is prescribed for severe invasion with neurological disorders, myocarditis, IHD, pulmonary insufficiency. Glucocorticosteroids are used: usually prednisolone in a daily dose of 20-60 mg orally( according to indications - and up to 80 mg) for 5-7 days, then the dose of the hormone is reduced with cancellation of the drug after 1-1,5 weeks. Due to the fact that hormones can prolong the period and increase the amount of trichinella production in the intestine, it is recommended to prescribe anthelminthic drugs( vermox or albendazole) during the entire period of corticosteroids and several days after their cancellation. Danger is also possible ulcerative lesions in the intestine in combination with violations in the venous system. The risk of ulcerative action, hormones increases dramatically, especially with the simultaneous administration of anti-inflammatory drugs( indo-metacin, diclofenac, etc.).In these cases, for the prevention of ulcerative lesions in the gastrointestinal tract, it is recommended to use proton pump inhibitors( omeprazole, etc.).In patients with severe trichinosis with the presence of generalized edema due to accelerated protein breakdown and lack of protein, intravenous therapy with the introduction of detoxification agents and preparations for parenteral protein nutrition is recommended.

Forecast. In case of mild and moderate disease, timely and adequate treatment, the prognosis is favorable. In severe cases and with the development of complications, the prognosis is serious. Children tolerate the disease usually easier than adults, but they also have severe fatalities.

Prevention .The basis for the prevention of trichinellosis is the provision of veterinary and sanitary supervision and sanitary and educational work. To prevent people's diseases, the most important are the mandatory veterinary examination of meat and extensive propagation through the media of information about helminth diseases and the ways of its spread.

Diagnosis of VHF is invaluable in trichinosis, especially for differentiated diagnosis between cancer and tumors formed by larvae. But when a person has good immunity, high reserves of adaptation, high anti-cancer resistance( immunity), good health, and at the same time trichinella larvae are tested in a tumor, then, you know, this compaction can not be a cancer tumor in any way. Thus, with trichinellosis, the diagnosis "cancer" is removed. Complex treatment: HRT, albendazole, cleansing of the body - leads to positive results until the disappearance of tumor trichinella and the reduction of uterine fibroids, as well as the termination of uterine bleeding.

Trichinellar breast tumors respond well to resonance frequencies, that is, frequencies of Trichinella itself in a wide range. But it is impossible to treat only HRT: the protective solid capsule of trichinella protects them so much that they, weakened, as if asleep, under adverse conditions( stress, emotions, other diseases) can grow stronger and start increasing the tumor again. Therefore, it is necessary to kill them in a complex way: HRT, albendazole, purification of the body.

The capsule itself( cocoon) decreases very slowly. This is very disturbing to the patient. In such cases, after the elimination of trichinella as living creatures, it is possible to make the operative removal of this formation( cocoon) with the preservation of the mammary glands and lymph nodes.

Remember: if you even cut out the tumor whole, live Trichinella will have time to send information( SOS) to other of their counterparts, and they are activated and will try to spread faster, strengthen, up to, the brain.

When removing the tumor with the opening of the capsule - larvae's dwelling - they escape through the blood vessels and become free-floating, freely moving through tissues, organs. And then they also hit new vital organs.

In some patients operated with a diagnosis of "breast cancer", with surviving live migratory Trichinella under the influence of resonant antitrichinella frequency therapy, sensitivity to their movements is manifested. When cries like "oh, oh, they ran to another place" at first thought that the patients, they say, "the roof went".But after the fifth such case, they began to check this phenomenon at the WFD.And it turned out that the patients were right: where we had influenced HRT, Trichinella was no longer there. And where the patients indicated, Trichinella were detected.

It is very pleasant to remove the diagnosis of "cancer" and save women life and mammary glands.

Teniarinhoz - helminthic invasion caused by a bull chain that can parasitize a person, being not only in the sexually mature, but also in the larval stage, causes the disease of cysticercosis.

Infection occurs when raw or semi-raw beef finnish meat is eaten( shish kebab, minced meat, sirloin from meat).From finn - larvae of bovine tapeworm, caught in the small intestine, - the head is turned out and attached to the intestinal wall, in 3 months a mature worm is formed. Its length reaches 5-6 m and more, the head( 1-2 mm) is equipped with 4 suckers, the cervix passes into the body containing up to 1000-2000 proglottids( segments), each of which can include up to 150 thousand eggs.

The scheme of infection with a bull chain is as follows.

1. End segments are destroyed or actively creep through the anus;the allocation of eggs with feces leads to massive contamination of livestock, pasture, fodder, latrines, land.

2. With contaminated feed eggs enter the body of cattle, settle in muscles and turn into larvae - Finns.

3. Finns with a diameter of up to 0.5 mm contain the head of a mature worm. In the body of animals can exist for 1-2 years.

4. A person becomes infected with Finns by eating unprocessed meat.

Bovine tapeworm has a mechanical and toxic-allergic effect on the human body.

Other clinical manifestations: intestinal spasms, oppression of gastric juice secretion, nausea, vomiting, dizziness, irritability, anemia, laryngospasm, Miniera syndrome, attacks of pain, characteristic of appendicitis, cholecystitis, pancreatitis.

Diagnosis of the shadows is based on the detection in feces of patients with mature segments of bovine tapeworm.

Treatment with orthodox medicine. Assign prazikvantel 5 mg / kg body weight once 3 courses with an interval of 7 days.

It's very difficult to fight with a chain. Its head is so strongly attached to the intestinal wall that it is sometimes impossible to tear it apart. He would rather throw off his body, consisting of joints and eggs, than relax his suckers. Even HRT can not make him do it. Under the action of RFT, the tartar rejects its body.

Therefore, in order to relax it, we carry out the tjubazh not with one oil, but in half with vodka or cognac - to whom that is tested. But even this method does not guarantee a 100% victory. I have to repeat the procedure.

Very valuable is the fact that with the help of an inductor for WFD, you can accurately locate the head of the chain and then destroy it with frequencies.

With this treatment, getting rid of this monster is much more successful.

Teniosis is a disease caused by a pork chain( synonym: tapeworm armed) of the shadow of the saltium - a flat worm, related to the diodes.

Causes of the disease. The parasite has a length of 1.5-2 m and the number of segments from 800 to 1000. The scolex( head) is about 1-2 mm in diameter, has 4 cruciformly located suckers and proboscis on which there is a double "crown" of 22-32 hooks. The segments in the anterior part are shorter than the width, and in mature segments at the end of the strobila, the length always exceeds the width. Eggs practically do not differ from eggs of bovine tapeworm.

Structure and spread of the disease. The spread of the shadow is widespread. The development of the pork chain takes place with a change of owners. The final host is a person, in between - a pig, which becomes infected when eating segments or eggs of the worm.

The source of human infection by the shadow is not enough decontaminated meat of pigs and less often - wild boars. The greatest danger is pork, which has not undergone veterinary control.

Mechanism of development and ways of spreading the disease. In the pig's stomach, the egg-clad egg-clad egg shell collapses, the released oncospheres actively penetrate the circulatory system of the stomach or intestine and flow through the body. After 24-72 hours oncospheres settle mainly in the intermuscular connective tissue, where in 2 months they become a Finn( cysticerci).

Then with food get into a person where cysticercises turn into adult parasites in the intestines. Under the action of digestive juice and bile, the head is turned out, which is attached to the mucosa by means of suction cups. The parasite begins to grow, forming the segments, and after 2-2.5 months reaches maturity. Grows for several years.

In the pathogenesis of teniosis, a significant role is played by toxicoallergic reactions, mechanical irritation of the mucous membrane with suckers and hooks, absorption of the host nutrients.

Symptoms and course of .In patients, there are disorders of bowel function, weakness, dizziness. There are nausea, vomiting, abdominal pain, periodic headaches, sleep disturbances. In some cases, neurologic symptoms are associated with the possible presence of cysticercosis of the brain. Sometimes, shadows are accompanied by a decrease in iron in the body, anemia. Often, clinical manifestations are absent.

Diagnosis of taeniosis is based on the detection in feces of patients with mature segments of the pork chain, which are separated in groups of 5-6, more rarely - singly. Active segments do not have segments.

Treatment with orthodox medicine. Therapy is carried out prazikvantelom - 5 mg / kg body weight once.

The forecast is generally favorable. Prevention consists in a mass survey of the population for the presence of helminth diseases, treatment of invasive diseases, strengthening of veterinary control over the slaughter of pigs and pork meat.

People come to our Center with all kinds of diagnoses, ranging from anemia, vegetovascular dystonia, dysbacteriosis, gastritis, enteritis to psychopathy. But the cause of these diseases remains unrecognized.

When diagnosing WFD, the cause of all these symptoms is clearly identified - shadow. Well, to treat after diagnosis is already easy: resonance frequency therapy( HRT), praziquantel and purification of the body. Surprisingly, after treatment, even on a sparse vegetable diet, anemia quickly disappears and a vigorous, well-being is restored.

Cysticercosis is a disease associated with the parasitization of the larval stage of the pork-cysticerca chain in the human tissues, or finn.

Causes of the disease. In eggs enclosed in segments of pig or bovine tapeworm, there is a fully formed embryo( oncosphere).The development of cysticercosis in humans is possible when oncospheres enter the stomach through the mouth or when back peristalsis from the intestine to the stomach. Further development occurs, as in the pig's body.

Mechanism of development and ways of spreading the disease. It is believed that the life expectancy of a cysticerc in a human usually reaches 3-10 years. In the development of the larva, the stages of a viable parasite, dying and completely dead, are distinguished. In the last period, the greatest changes in the organs are observed. The diameter of the cysticercles is usually 5-8 mm, around it a reactive connective tissue capsule is formed. Parasitizing cysticerci is possible in various organs and tissues( muscles, lungs, brain, bones, eyes, subcutaneous tissue, etc.).In the brain tissue surrounding the capsule, endarteritis and infiltration of tissues around the vessels predominantly by plasma cells develop. These changes sometimes give a picture of encephalitis. Cysticerci can parasitize in cavities( in the ventricles of the brain, anterior chamber of the eye or vitreous body).In soft brain tissues, sometimes forms a branched( racemose) form of cysticerca up to 25 cm in length. The stage of withering of the parasite is characterized by its swelling and melting and the most pronounced toxic effect in this period. The death of cysticerci is often accompanied by subsequent calcification.

Symptoms and course. Clinical manifestations depend on the location of the parasites and their number, the stage of the process and the characteristics of the patient's body.

Cysticercosis of the brain is characterized by instability of clinical symptoms;the phenomena of irritation of brain structures prevail over the phenomena of loss of functions. The main symptoms are paroxysmal headaches, nausea, vomiting, epileptiform seizures. Sometimes a disturbance of the psyche is noted in the form of hallucinatory and weak-minded states, which can suddenly disappear and reappear.

Cysticercosis of the ventricles of the brain is accompanied by manifestations of intracranial hypertension, their occurrence requires urgent surgical intervention.

Cysticercosis of the eye causes visual disturbances and often blindness. Lesions of muscles, subcutaneous tissue and skin often occur without complaints.

The diagnosis is based on the presence of clinical manifestations, biopsies of subcutaneous nodes, data of X-ray examination.

Differential diagnosis is more often performed with tumors, inflammatory diseases of organs and echinococcus.

Treatment with orthodox medicine. From therapeutic measures, the surgical method in combination with anti-inflammatory therapy is the only radical method of treatment. However, the possibilities of surgical intervention are limited and depend on the number of parasites and their location. With multiple cysticercosis, X-ray therapy is indicated. Sometimes it is possible to obtain an effect from the use of chloridine in combination with sulfanilamide preparations. There were reports of the effectiveness( in 84%) of the use of albendazole 15 mg / kg / day for 8 days( prazikvantel in the same doses is effective in 62%).Anthelminthic drugs are contraindicated in the acute period of cysticercoid encephalitis, and prazikvantel is contraindicated in case of eye damage.

The prognosis depends on the location and amount of parasites in a particular organ.

Prevention and control measures are the same as with shadow.

If the cysticerci have affected the fetal brain, then the child is born either dead or ugly.

Therefore, before conceiving a child, it is necessary to exclude the presence of a chain or to carry out prophylactic antihelminthic treatment of future parents.

In these cases, the role of vegetative resonance diagnostics of VRD and resonant frequency therapy( HRT) is invaluable.

Gimenolepidosis is a helminth disease that occurs with signs of primary lesion of the gastrointestinal tract.

Pathogen - dwarf catcher

Dwarf chain is a small cestode with a length of 15-30 mm, a width of 0.55-0.70 mm with a very small head, equipped with 4 suckers and a short proboscis with a whisk of 20-24 small hooks. The body of the chain consists of a thin neck and a large number( up to 200) of segments. Hermaphrodite.

Structure and distribution of the disease. Hymenolepiasis is widespread. The source of the invasion is a person.

The mechanism of development and ways of spreading the disease are largely determined by the cycle of parasite development. The cycle of development of the dwarf chain is characterized by the successive development of the larval and adult stages in the human body. Thus, the person for this worm is both an intermediate and a main host( Figure 39).The chain lives in the small intestine of a person. Eggs separated from the joint contain a formed embryo, are contagious and do not need ripening in the external environment. From the egg, which got into the mouth, and then into the human intestine, the oncophera is released, which is actively introduced into the villi of the small intestine. After 5-7 days, a larva develops from the oncosphere, a cysticircoid that destroys the villus, emerges into the lumen of the small intestine, attaches to its mucous membrane, and grows to an adherent adherent in 14-15 days.

Symptoms and course. Hymenolepiasis sometimes occurs asymptomatically, most patients are concerned about nausea, vomiting, heartburn, changes in appetite, and dull pain in the abdomen. The pain occurs in the form of daily attacks or with interruptions in a few days. Characteristic is an unstable liquid and rapid chair with an admixture of mucus.

As a result of prolonged diarrhea and loss of appetite, weight loss is observed. In children, hymenolepiasis takes place in a more pronounced and severe form. Drowsiness, exhaustion, nervous twitching and even epileptiform-type seizures in children can be a consequence of hymenolepiasis.

There is usually no marked or characteristic blood change in hymenolepiasis. Sometimes there is a moderate decrease in hemoglobin and a slight increase in eosinophils( blood cells).

Diagnosis. The diagnosis is based on the detection of eggs in the bowel movement. Eggs of the dwarf chain are allocated cyclically, so with negative results it is necessary to repeat the tests, and also to combine with the study of the smear from the anus.

Treatment with orthodox medicine. Features of biology of the parasite, the possibility of self-infection require persistent and systematic treatment, the use of not only anthelmintics, but also symptomatic therapy in conjunction with a set of preventive measures. To get rid of helminths, biltricidum is used - 10-25 mg / kg of body weight orally once. The drug is not recommended for children of the first two years of life, contraindicated in pregnancy.

The prognosis for hymenolepiasis is favorable( in case of causal therapy).

Prevention. The thorough conduct of sanitary and hygienic measures significantly reduces the incidence of hymenolopidosis. It is necessary to strictly monitor the cleanliness of the body, dwelling, office space, especially children's institutions.

Pathogen - rat catheter

Rat chain - hymenolepis diminuta - more formidable and larger in comparison with dwarfish - reaches 60 cm( Figure 40).For the first time these worms were discovered in 1851 by Bilharz in the ileum of a boy who died of meningitis.

Structure and spread of the disease. Rat tartar is found in all climatic zones.

Infection occurs when a person accidentally swallows a particle: a caterpillar of a mill fire, a flour meal, some cockroaches, feces of rats, mice and other rodents;sometimes feces of dogs, cats( eating rats, mice), monkeys.

Adults are rarely infected. Infection occurs through the mouth, through contaminated hands, food, household items, toys.

The difference between the rat chain and the dwarf chain is as follows.

1. Infection does not occur from a person, that is, rat tapeworm is a zoonotic( from animals) worm that penetrates through the mouth.

2. There is no repeated self-infection of your own eggs with a chain.

3. The larvae of rat tartar can spread rapidly through the nervous system( brain and spinal cord).

4. Lead to meningitis, encephalitis, paresis and paralysis.

In families where hand-held rats are available, mice usually detect ratshopen often in children. Parents are surprised at allergies in children, their irritability, memory loss, academic performance, lethargy and do not understand that the culprits of these manifestations can be their favorite pets.

Diphyllobothriasis is caused by the largest worm, which only invades the human body. This diphyllobothrium is latum - a wide ribbon is the most common human parasite.

In humans, it causes intoxication and anemia.

The final hosts of the parasite are humans, bears, dogs, fish-eating birds, cats. Diphyllobothriasis is affected by people who eat seafood and light-salted fish, fresh fish in Korean.

The infectiousness period for others comes in 1.5 months after infection and lasts throughout the parasite's stay in the body( up to 10-30 years).The spread of the disease is facilitated by the contamination of water bodies by feces as a result of the discharge of untreated wastewater into them.

Diphyllobothrium - as already mentioned, one of the largest parasites of man, reaches a length of 10 m and more, the head is 3-5 mm, oblong, oblate, has two suction cracks( botry), the body consists of 3000-4000 proglottids( segments).

Adult individuals parasitize the small intestine;eggs are excreted with feces and for further development must enter a freshwater body of water.

In the water from the egg, a larva is covered with cilia, which is swallowed by freshwater Cyclops, and the latter by fish. In the body of fish, the larvae pass through the next stage of development - the plerocercoid and penetrate into the muscles, caviar and internal organs.

A person becomes infected when using poorly processed fish, salted caviar and stroganina.

The plerocercoids are attached by the botryas to the wall of the small intestine and after 2 months they grow into an adult. In the human body, the ribbon lives 10-30 years or more.

Clinical manifestations are usually poorly expressed: patients note increased fatigue, dizziness, nausea and abdominal pain. In severe cases, it is possible to observe the development of anemia by the type of malignant anemia. Addison-Birmer.

With such malignant diagnoses, the patient comes to us. And when after resonant frequency therapy and purification of the organism, creatures of 4 to 6 m in size emerge from them, then all malignancy in a person disappears. People become healthy, workable.

The diphyllobothrium makes a person eat a lot, but the person remains thin and thin, as they say, "not in the horse's forage".Very often women complain about children and husbands: "Eats for three, and everything is thin."This is because tests on the eggs of the helminth are necessary to take several times to obtain a positive result.

Treatment with orthodox medicine is complex. Diphyllobotriums are so strongly attached to the wall of the intestine that it is not possible to tear them apart.

When taking a drug of nikrosalide, diphyllobothrium resets its body, and the head remains and begins to grow again.

It is necessary to cope with it in a complex way: resonant-frequency therapy, the inductor should be placed precisely on the area of ​​its head( previously tested this place), nikrozalid or albendazole, vermox( which of them is tested), then intestinal cleansing, tjubazh with oil + vodka or cognac, lemon juice and again irrigation of the intestine. Only so get these 2-6-meter monsters.

Recipe. Oil 50 ml + vodka or cognac 30 ml are mixed and drunk at night. Drink 30-50 ml of lemon or any sour juice. Well before that, drink 25 g of magnesium sulfate or use laxative herbs throughout the day.

Strongyloidosis is a helminth disease caused by intestinal ugliness. In the early stage, the predominant symptoms are allergic to the skin and lungs, in the late stage, the defeat of the digestive system. Strongyloidosis was first described in 1876 by Norman under the name "Cochin diarrhea".

Сронгилоидоз it is distributed mainly in the countries with a tropical and subtropical climate. Single cases are recorded in the temperate climatic zone. Strongyloidosis occurs in the Transcaucasian countries, Moldova, Ukraine, Central Asia, Russia - in some Southern and Central regions.

Causes of the disease. The causative agent of Stron-Hyloidosis is a round worm - intestinal uger. The male is 0.7 mm long, 0.04-0.06 mm wide. The female is 2.2 mm long, 0.03-0.7 mm wide. Eggs are transparent, oval in shape, measuring 0.05 x 0.03 mm. The development of parasites proceeds with the replacement of free-living and parasitic generations. Sexually mature females of the parasitic generation are localized in the human body in the thickness of the mucous membrane of the duodenum, penetrate into the stomach, mucous membrane of the small intestine, pancreatic and bile ducts with intensive invasion. Fertilized females lay eggs, from which larvae come out, reaching a length of 0.2-0.3 mm. Larvae are excreted with feces into the external environment, where they turn either into filarial larvae, or into free-living mature males and females capable of laying eggs.

Filarium larvae are able to invade a person, penetrating through the skin or through the mouth with water and food. When infected through the skin, the larvae migrate, similar to the larvae of ascarids and acetylostomide. In the human body, they make their way through the blood vessels, enter the small circle of circulation, enter the lungs, where they give rise to a new generation of parasitic females and males. Hence, young specimens of intestinal blackheads reach the intestine through the respiratory tract, pharynx, esophagus.

Structure and spread of the disease. The source of infestation is a person with strongyloidiasis. Infection comes from the soil. Particularly favorable conditions are in countries with hot and humid climates, as well as in hot mines and tunnels that are hot with high temperatures.

Symptoms and course. In the early migration stage( up to 10 days) strongyloidosis, fever, itching, hives or papular eruptions, local edema, eosinophilic infiltrates appear in the lungs.

In the late phase of the invasion, when the worms reach puberty, sometimes severe manifestations can be observed with a fatal outcome. In mild cases of the disease, nausea and dull pain in epigastrium are noted. The chair, as a rule, is not changed, constipation or alternating constipation with mild diarrhea is possible. Sometimes leading is an allergic syndrome. In severe manifestations, nausea is often accompanied by vomiting, acute pain occurs in the epigastrium or throughout the abdomen. Periodically there are diarrhea - up to 5-7 times a day. The liver in some patients is enlarged and compacted. In peripheral blood, most patients experience eosinophilia up to 70-80%, with prolonged invasions, secondary anemia, usually mild, occurs. In severe forms of strongyloidiasis, diarrhea becomes permanent, the stool gets a putrefactive odor and contains a lot of undigested food. There comes dehydration, severe secondary anemia, exhaustion. On the part of the nervous system, headache, dizziness, increased mental fatigue, neurasthenic and psychasthenic syndromes are noted. There are symptoms of duodenitis, enterocolitis, less often - angiocholitis and hepatitis. In the absence of treatment, the helminth disease acquires a prolonged chronic course. Possible lung damage.

Diagnosis and differential diagnosis. Parasitological diagnosis is established by the detection of larvae( less frequently eggs) in duodenal contents and in feces treated by the method of Berman, in the migratory stage it is sometimes possible to identify larvae and sexually mature parasites in sputum. All patients with high eosinophilia of blood should be examined for strongyloidiasis.

Treatment with orthodox medicine .The therapy is carried out with ivermectin, levamisole, thiabendazole. Levamisol( decaris, askaridol, shenizol) is prescribed for the night in one dose - 120-150 mg to an adult, children - 2.5 mg / kg body weight. A second course of treatment - a week.

Tiabendazole( vermox) is indicated for migrating forms of strongyloidiasis. Assign a dose of 25 mg / kg body weight every 12 hours for 2 days after eating( with a body weight of more than 54 kg, a single dose of 1.5 g).The maximum daily dose should not exceed 3 g. The second course of treatment can be prescribed according to the indications in a week.

Ivermectin( metizane) is prescribed at 200 mg / kg / day for 2 days.

The prognosis is usually favorable. According to Dr. Clark( USA), this type of worms is always found in cancer patients. But this is in their climate. In Russia, according to our Center, in oncological patients more common schistosomiasis, echinococcosis, opisthorchiasis.

Fascioliasis is a helminth disease caused by fasciolias( synonym: fluke), with a predominant lesion of the bile excretory system.

Causes of the disease. The causative agents of fascioliasis are two types of worms - fasciola hepatitis( hepatic) and fasciola gigantica. Fasciola hepatic has a foliate shape: length 20-30 mm, width 8-12 mm. The fasciola of the giant forms 33-76 mm in length, 5-12 mm in width. The final masters of fasciolae are man and numerous herbivores( large and small cattle, odd-toed ungulates, etc.).Under normal conditions, the worm is localized in the bile excretory system, occasionally in other organs. Intermediate hosts are various species of freshwater mollusks, especially small pond snails. The lifespan of fasciolae in the human body is 3-5 years or more.

Structure and spread of the disease. The main source of infestation is herbivorous animals, sick with fascioliasis. The role of the sick person is insignificant. Eggs of the worm are released into the external environment with faeces of animals. When they get into water or into moist soil, larvae miracidia develop after 4-6 weeks. In the water, the meracidia leave the eggs and are introduced into the intermediate hosts - the mollusks, where they turn into cercariae. Infection of animals occurs when swallowing larvae with water or eating grasses growing in water bodies with standing and slowly flowing water. Infection of a person occurs when drinking raw water and eating food plants found in water bodies and in wet places. Fascioliasis is quite widespread.

Mechanism of development and ways of spreading the disease. Fasciola larvae, caught with food or water in the gastrointestinal tract, hatch from the surrounding shells and penetrate the biliary system, and sometimes into other tissues. Perhaps this migration takes place through blood vessels. The larvae, penetrating into the circulatory system of the portal vein, penetrate the abdominal cavity through the peritoneum, migrate to the liver and through its glisson capsule penetrate the hepatic parenchyma, then into the bile ducts, where the sexual cycle of their development ends in 3-4 months. During migration, young and adult fasciolae cause mechanical damage to tissues, and sometimes can cause complete mechanical blockage of the bile duct. The products of the vital activity of the worm cause toxic-allergic reactions, which are most clearly manifested in the early period of the disease.

Toxico-allergic reactions cause irritations of the nerve endings of the bile ducts, which affects the functional state of the gastrointestinal tract and pancreas. Disorders of normal circulation of bile along the ducts create favorable conditions for the onset of secondary infection. In the liver parenchyma, micronecroses and microabscesses are sometimes found. In the later period of the disease, lumen enlargement, thickening of the walls and adenomatous growth of the epithelium of the bile ducts occur, and sometimes purulent angio-cholangitis develops. Violation of the passage of bile promotes the formation of stones in the gallbladder.

Symptoms and course. The incubation period with fascioliasis lasts 1-8 weeks. The disease begins with the appearance of weakness, malaise, headache, decreased appetite, itching of the skin. Body temperature is somewhat elevated, laxative, wavy. In severe cases, it can rise to 39-40 ° C.At this point, there may be hives, a slight icterus sclera. Begin to disturb pain in the left hypochondrium, in the right hypochondrium, nausea and vomiting. The liver is enlarged. By consistency, it becomes dense and painful when palpated. The left side of the liver often increases, which leads to swelling of the left hypochondrium. Pain in the liver is often paroxysmal. After cessation of pain, the size of the liver is somewhat reduced. Spleen rarely increases. In the study of blood marked leukocytosis( 18-20 x 109 / L) and eosinophilia( up to 85%).The body temperature gradually decreases to normal, and the disease turns into a chronic form. At the same time leading dyspeptic symptoms are diarrheal symptoms: pain in the left hypochondrium, often having a paroxysmal character, a decrease in appetite, bloating, rumbling, etc. There is an increase in body temperature to 38 ° C and above. In the future, jaundice develops. The duration of attacks is from several hours to 7-8 days at short intervals. The liver increases, protruding from under the edge of the costal arch by 2-5 cm, with palpation dense and painful, with a flat surface. Leukocytosis becomes insignificant, and eosinophilia decreases( 7-10%).In the long course of the disease, diarrhea, macrocytic anemia, hepatitis and severe exhaustion appear, which can lead to death. The disease can occur in mild, moderate and severe forms. Early and late clinical manifestations of the disease, as well as the degree of its severity, depend on the intensity of the biliary excretion of the bile excretory system, secondary infection, eating disorders, vitaminization and the influence of other factors.

Diagnosis and differential diagnosis of .In the early stages of the disease, diagnostics cause great difficulties, since at that time the eggs of the worm can not be detected. In the patient's body, they reach puberty only after 3-4 months. It should be distinguished from trichinosis, the signs of which are puffiness of the face, pain in the muscles of the extremities. These symptoms are absent in fascioliasis. During puberty fasciol, diagnosis does not cause great difficulties, due to the fact that typical eggs of the worm are found in feces and duodenal contents. With a weak invasion in the study of stool, Goryachev's deposition method is used. When eating liver-infected fasciola of cattle in feces, transit eggs can be found. In these cases, a second analysis is necessary 2 weeks after exclusion from the liver.

Treatment with orthodox medicine. The fascioliasis is treated in the same way as opisthorchiasis, prazikvantel and albendazole. However, both of them showed insufficient effectiveness. Triklabendazole is currently being tested. In pharmacies it is not yet.

Forecast. With mild forms of flow of the fascioliasis and timely treatment, the outcome is favorable. With moderate and severe forms of the flow, residual disturbances in the function of the bile-excreting system can be observed.

Preventive measures and measures in the outbreak. Water from non-water reservoirs is used for drinking only boiled or filtered through tissue. Plants, spread in moist places, eaten in boiled form or scalded them with boiling water. The struggle against mollusks is carried out by melioration and extermination by chemical substances( copper vitriol, lime, etc.).

Often reveal fasciolae hepatitis with chronic course of parasitic hepatitis, cholangitis, cholecystitis, pancreatitis.

Precise diagnosis of hepatitis fasciola allows directing resonant frequencies through inducers directly to the sites of localization of parasites in organs and even locally, in certain areas of one organ, for example, the liver.

Further decoctions of laxative and antiparasitic herbs( wormwood, tansy, dandelion root, buckthorn bark, flax seed, etc.) cause them to separate from the organs and cleanse, tuffle the liver and wash them from the body.

Without resonant frequency therapy( HRT), it is very difficult to derive them.

Opisthorchiasis, a helminth disease caused by opisthorchis, is characterized by a lesion of the hepatic bile( hepatobiliary) system and pancreas.

In man, opisthorchiasis was first described in 1891 by KN Vinogradov.

Causes of the disease. Pathogen - trematodes of the liver( or flukes, flukes) - a flat worm 5-20 mm long, 1-4 mm wide. The eggs released by the opisthorchus are small, measuring up to 0.026-0.01 mm. They are also called cat or Siberian fluke.

The situation is complicated by the fact that opisthorchis are hermaphrodites.

The final owners of opisthores, except humans, can be many animals - rodents, predators, etc. But man creates opisthorchis best conditions for parasitism: they can freely reproduce up to tens of thousands and live in one organism up to 40 years. In the human body, the opisthorchis began to parasitize relatively recently - about 15-20 thousand years ago.

Opisthorchiasis, mainly affecting the hepatobiliary system and pancreas and characterized by prolonged course and frequent exacerbations, promotes the emergence of primary cancer of the liver and pancreas.

Mechanism of development and ways of spreading the disease. Larvae of opisthorchis enter the intestine of the human with their edible fish and come out from the surrounding shells and penetrate the liver, gall bladder and pancreas into the liver, gallbladder and pancreas where they reach puberty after 2 weeks and begin laying eggs in a month.

As a result of the isolation of the products of their metabolism by the worms, allergic reactions occur( especially pronounced in the early phase of the disease).The glistens also have a pathogenic mechanical effect, which consists in damaging the walls of the biliary and pancreatic ducts and the gall bladder with suckers and shi-peaks covering the surface of the body of the worm. The accumulation of parasites causes a slowing of the current of the bile and secretion of the pancreas, neural-reflex signals by stimulation of the nerve elements of the ducts, resulting in nerve pathological impulses, primarily transmitted to the stomach and duodenum. The activity of worms leads to dyskinesia of the bile ducts, accumulation of parasites, eggs, cells of the lowered epithelium in the biliary tract, and the cessation of the flow of bile. This creates "favorable" conditions for the formation of stones and for the attachment of secondary infection of the biliary tract, as well as the glandular proliferation of the epithelium of the biliary and pancreatic ducts, which should be considered as a precancerous condition.

In the acute stage of opisthorchiasis, differential diagnosis with viral hepatitis is often necessary. The main symptoms that distinguish opisthorchiasis from viral hepatitis are: acute onset, high long-term fever, a short period of relatively mild jaundice, pain in the right hypochondrium and an increase in leukocytes, eosinophils in the blood. The acute stage with clinical manifestations usually develops in persons who came to the outbreak from non-endemic areas in the opisthorchiasis. In the local population, the disease usually has a primary chronic course. In particular, acute opisthorchiasis is practically not observed in the indigenous peoples of the North( Khanty, Mansi), whose involvement in chronic foci of opisthorchiasis reaches 100% in some foci.

The clinical picture of chronic opisthorchiasis is manifested primarily by the symptom complex, characteristic of chronic hepatitis, cholecystitis, duodenitis and pancreatitis. The leading symptoms are pain in the upper abdomen, mainly in the right upper quadrant, nausea, poor tolerance of fatty food, dryness and bitterness in the mouth, etc. Often with prolonged course, depression develops with anxiety-hypochondriac syndrome, "glistophobia", "withdrawal to illness".In some cases, the course of the disease can be severe, with overlapping of the bile ducts, development of jaundice, recurrent cholangitis, liver abscesses, acute pancreatitis and biliary peritonitis( inflammation of the peritoneum).

Especially important is the role of opisthorchiasis in the origin of cancer. Numerous studies have shown the association of opisthorchiasis with tumors of the hepatic-biliary system and, above all, with cholangiocarcinoma( bile duct cancer).In Russia, the number of cancer and liver diseases is the most significant in the Tyumen region, where the level of opisthorchiasis is also very high. There are observations that indicate the important influence of these worms on the development of cancer of the pancreas and gallbladder.

Diagnosis is extremely difficult. Only with repeated duodenal probing can be obtained in the contents of opisthorchis. Puncture of the liver will not be drawn into the syringe of opisthorchis. Trial opening of the abdominal wall( laporotomy) with excision of pieces of liver, given in histology, ends in a standard way: micron thickness sections are made, naturally, atypical cells are found and diagnosed as "cancer".Yes, you need to know exactly where to take a piece of research and how to look at it.

Prevention. Fish decontamination is achieved by heat treatment, freezing, smoking, salting in accordance with the developed recommendations:

• freezing, at least 7 hours at a temperature in the fish body - 40 ° C, 32 hours at 28 ° C;

• pickling with a density of 1.2 at + 2 ° C, the duration of salting - from 10 to 40 days, depending on the weight of the fish;

• cooking - at least 20 minutes from the time of boiling.

Treatment with orthodox medicine. Praziquantel( biltricide) 40-75 mg per 1 kg of body weight in 2-3 divided doses 7 days. Break - 7 days. It is recommended to conduct 3 such courses.

1. Birch tar. Start with one drop. Drip on sugar and rassasyvat, squeezed a small amount of water 1 time per day. To diabetics or having predisposition to this, the tar drips onto the bread and dissolves. Each day, add one drop and bring to 5-8 drops( according to the state of health).Then continue to 5-8 drops daily for a month. Take a break for 1 month and repeat the course again.

2. Purified kerosene. It is necessary that kerosene be distilled at a temperature of 100-160 C!Begin with one drop of sugar or bread, adding 1 drop per day to 15 drops per day( for adults).13 days to take, 13 days - a break, then take another 13 days.

For prevention, you can take 1 teaspoon per day on an empty stomach for 13 days twice a year - in spring and in autumn.

3. Pharmacy medicine - prazikvantel.1 tablet 2 times a day for 10 days.10 days break. These methods to hold 3 times.

Foci of schistosomiasis in antiquity existed in the basin of the Nile. This is evidenced by 4 Egyptian papyrus found by archaeologists( Ebere, Berlin, Hirst, London), dating from 1550-1225 BC.e. They describe the disease "aaa" with the following symptoms: the presence of worms in the body, an admixture of blood in the urine, damage to the rectum, diarrhea.

In 1910, paleontologist Ruffer conducted research on the Egyptian mummy of the 20th dynasty of the pharaohs( 1200-1130 BC).In microscopic sections of the kidneys, a large number of eggs of the urogenital schistosome were found.

Historians-Arabs testify that among men of Ancient Egypt there was a disease in which they "menstruate, like women."The disease was called "Egyptian hematuria".From Egypt, the disease spread to other parts of Africa, spread to Asia, America and China.

In China, more than 2,000 years ago, Japanese schistosomatosis( eggs found in the mummified corpse of a woman) was common.

Also a large foci of schistosomatosis was found in the basin of the Tigris and Euphrates rivers.

Chronic polyposis colitis arising from schistosomiasis is classified as a precancerous disease that can lead to colon cancer. "

Scientists De Leon and Tavera in 1968 described two cases of lymphosarcoma arising from Japanese schistosomiasis. Also, schistosomatous granulomas in the brain resemble a tumor.

The lack of measures to protect the population during the transformation of nature leads to the spread of parasites. For example, the creation of reservoirs in Senegal led to a massive infection with schistosomiasis( up to 70% of infected).

Schistosomes are found in strata of the earth, dating from about 1-1.5 million years.

A person easily infects parasites by contact with water. In the tropics, man is almost the sole owner of schistosomes.

Polyps, polyposis, endometritis, endometriosis, recurrent sand, kidney stones, recurrent pyelonephritis, cystitis, prostatitis, tumors, fistula in the coccyx region - all this may result from schistosomal injury.

Schistosomes - worms of different sexes. Each pair can produce from 300 to 3000 eggs per day. Eggs have thorns and are able to pierce and pass not only the tissues of the bladder, uterus, liver, spleen, lungs, stomach, intestines, but also through the walls of the blood and lymphatic vessels. In places of accumulation of parasites and their eggs, the lumen of blood vessels and ducts is clogged, which leads to the formation of varicose veins, cysts, and tumors.

Young parasites go from skin and intestines to the lymph and blood vessels through the lungs and the heart to get into the liver, and only in the liver they grow to mature individuals, where they then mate. Further, depending on the species, migrate against the flow of blood into the veins of the bladder, the kidneys, the collar and mesenteric veins, where eggs are laid. Sometimes they settle in colonies in narrow places and form varicose enlargements, nodes of veins.

The life span of schistosomes in the human body is dozens of years. When you leave the person for further infectious activity, the eggs of the schistosomes, thanks to the thorns, can independently move through the walls of the vessels of any organs - into the bladder or into the intestines - and then be excreted in the urine or with feces.

And we can penetrate into us when bathing from 1500 to 4000 pieces of cercariae with schistosomes. The reservoir of infection can be either a person or any animals, especially domestic animals.

Intermediate hosts of schistosomes are freshwater mollusks. Every day the body of the infected mollusk leaves up to 1500-4000 or more cercariae, and as a result, during the life of the mollusk - up to several hundred thousand invasive larvae of schistosomes.

A person becomes infected with schistosomes when bathing, washing clothes, working in contaminated water bodies, agricultural work on irrigated lands, during religious rites and other contacts with contaminated water.

Both natural and man-made reservoirs serve as a source of infection. At present, the role of irrigation( the creation of new reservoirs) in the epidemiology of schistosomatosis has increased. The emergence of new hotbeds is also facilitated by the intensification of population migration, related to the economic development of new territories, tourism, and pilgrimage. All people are susceptible to schistosomatosis, but the risk of infecting children is particularly high, since their games often occur near water bodies.

Female schistosomes retain their productive capacity for a long time. The pathogenic influence of parasites begins from the moment of penetration of cercariae through the skin. The effect of the secretion of the glands of migrating cercariae, the decay products of some of them are strong antigens and cause hypersensitivity reactions of immediate and delayed types. Clinically, this is manifested by a progressive papular itchy rash and is known as "scabies swimmers".Allergic reactions are the basis of the acute( "toxemic") phase of the disease, which develops during the completion of schistosome migration and the beginning of egg laying by parasites.

The basis of pathological changes in the chronic period of the disease is a set of inflammatory changes around the eggs of schistosomes - the formation of a specific cellular infiltrate - granuloma, followed by fibrosis, calcification and transformation into a tumor. Granulomatous reaction and fibrosis cause blood flow disorders in the organ wall, which is the cause of secondary dystrophic changes in the mucosa, the formation of ulcers. As a result of constant and prolonged stimulation of tissues by parasite eggs, products of their vital activity and decay, there may also be phenomena of growth and atrophy of the epithelium of the mucosa. In the bladder, in 85% of cases the main place of lesions associated with the deposition of eggs with schistosomes is the submucosal layer;the muscle layer is less likely to be affected. In the ureters, on the contrary, deeply located layers are more often affected.

In connection with the localization of the causative agent of intestinal schistosomatosis Mansoni and the accumulation of deposited eggs in the veins of the hemorrhoidal plexus and in the inferior mesenteric vein, the main pathological changes develop in the large intestine, mainly in its distal( terminal) sections. Schistosome lays not single eggs, but in groups, and they are more quickly subjected to calcification. With all forms of schistosomiasis, eggs are also introduced into other organs, primarily into the liver and lungs. The most severe liver damage leading to cirrhosis develops in Japanese and intestinal schistosomiasis. The ingress of eggs into the lungs leads to the development of obstructive-destructive arteritis, arteriovenous anastomoses, as a result of which hypertension of the small circulation circulates, resulting in a pulmonary heart( cor pulmonale).It is possible to introduce eggs of schistosomes into the dorsal and the brain. The severity of the clinical manifestations of the disease largely depends on the intensity of the invasion, that is, the number of eggs laid by the female parasites. At the same time, the granulomas around the eggs, the severity of fibrosis in the affected tissues are determined by the features of the host's immune response, in particular, by the level of antibodies, immune complexes, the activity of lymphocytes-suppresses, macrophages. Of particular importance are genetic factors, which, for example, affect the development of Simmers fibrosis( liver sclerosis - around the entrance to her hollow vein).

Schistosomiasis promotes carcinogenesis, and tumors of the genitourinary system and large intestine are relatively common in worm lesions. Carcinogenesis in schistosomiasis is explained by the development of fibrosis in the foci, epithelial metaplasia, immunosuppression, as well as the synergism of the action of schistosomes and exogenous and endogenous carcinogens.

Genitourinary schistosomiasis. In the clinical course, three stages are distinguished: acute, chronic and stage of outcomes.

Manifestations associated with the introduction of cercariae in the form of allergic dermatitis are rarely recorded in non-immune individuals.

Acute schistosomiasis may develop in 3-12 weeks of the latent period. Headaches, weakness, common soreness in the back and extremities, lack of appetite, increased body temperature, especially in the evening, often with chills and a strong sweat, there is a urticaria rash( unstable), the number of eosinophils increases to 50% or more. The liver and spleen increase. There are violations of the cardiovascular system and respiratory organs.

The earliest symptom of chronic urogenital schistosomatosis is hematuria, which is often terminal: at the end of urination, blood appears in the urine. There are pains in the suprapubic region and perineum. These symptoms are caused by the reaction of the tissues of the bladder and genital organs to the introduction of eggs with schistosomes. In later stages, cystitis due to a secondary infection may join. Cystoscopy on the mucous membrane of the bladder reveals granulomas - whitish-yellow formations the size of a pinhead, infiltrates, papillomatous growths, erosions, ulcers, "sand spots" - translucent through the thin mucous membrane of clusters of calcified eggs with schistosomes. Lesions of the ureters are accompanied by a narrowing of their lower third and the mouth, which creates conditions for stagnation of urine, the formation of stones, subsequently - pyelonephritis and hydronephrosis. There are also lesions of the genital organs: in men - fibrosis of spermatic cord, orchitis, prostatitis, in women - polyposis and ulceration of the mucous membranes of the vagina and cervix. In the late period, the formation of fistulas of the bladder and neoplasms of the urogenital system is possible. The defeat of the lungs and their arteries leads to hypertension of the small circle of blood circulation: the patients have shortness of breath, palpitations, signs of hypertrophy of the right ventricle of the heart.

Children, less often in adults, have cysts, abscesses, fistulas in the coccyx. As a rule, people affected by schistosomes develop urolithiasis. Children's bedwetting( enuresis).In men, acute, chronic prostatitis, prostate adenoma and concluding diagnosis - prostate cancer.

Particularly dangerous is liver damage.

The so-called hepatosplenomegalic( enlargement, compaction of the liver and spleen) form is characterized by a predominant liver damage with the outcome in the periportal( tissue around the vena cava) fibrosis and cirrhosis of the liver. Regardless of intestinal symptoms, patients note the appearance of a "tumor" in the upper half of the abdomen. Pains insignificant, disturbs feeling of gravity, discomfort. The liver is enlarged, dense, its surface is tuberous. With the development of portal hypertension, the veins of the esophagus and stomach are varicose, bleeding occurs due to their ruptures. The manifestation of decompensation of portal blood circulation is ascites. The spleen is enlarged constantly. In the future, decompensation of liver function occurs.

Treatment of orthodox medicine. The main treatment for patients with schistosomiasis at the present time is prazikvantel( biltricid, cesium, pikiton, azinoks), which is highly effective in all forms of worm infestation. The drug is prescribed in a dose of 75 mg per 1 kg of body weight of the patient in 3 divided doses after meals with an interval of 4-6 hours for one day. Adverse reactions are recorded quite often, but they are mild and short-lived: drowsiness, dizziness, headache, weakness, abdominal pain, sometimes rashes on the skin.

Forecast. In the acute period, the most severe forms of the course leading to death are recorded mainly with Japanese-Chinese schistosomiasis. In the chronic period with timely diagnosis and treatment, the prognosis is favorable for all forms. With the development of severe complications( hydronephrosis, liver cirrhosis with portal hypertension, etc.), the prognosis is serious.

But the most important thing is to avoid the word "cancer" in the diagnosis. And as you understand from the descriptions, with the defeat of any organs and systems, especially acute or in the diminishing stages, but with the formation of some enlargements or densities, ranging from papillomas, cysts, adenomas, hemangiomas, seals in the lungs, enlargement of the liver, spleen, pancreas, prostate, ovaries - with any such damage, you are always threatened with a diagnosis of cancer. Remember about schistosomiasis!

Cenular cerebral( synonym: pinworm) is a disease caused by the larval form of the price rus cerebralis of the ribbon worm of the multicep family.

Localization of larvae - head and less often spinal cord. The cestode ribbon stage is localized in the small intestine of definitive( permanent) hosts - dogs, sheep, cattle, yaks, camels, pigs. The main distributors are stray dogs.

Pathogens. Bubble( klairous) - censorus cerebralis( cerebralis) is light gray, filled with a transparent liquid, on its inner shell islands are 500 proto-scoices and more. The magnitude of the bladder in the brain depends on the degree of its development and often reaches the size of a chicken egg. More often in the brain, there is one, sometimes two blisters. A well-known veterinarian, doctor of medical sciences, professor NE Kosminkov believes that this type of bladder is often localized in the spinal cord.

In Central Asia, there is another kind of invasion - the price of muscle. It is characterized by the appearance in the various parts of the body of man and animals( subcutaneous tissue, surface and deep muscles) of tumor-like formations, at the opening of which they detect( if investigated) larvocysts of the centripetal type. Infection occurs when the segments of the eggs hit the intestine.

Mechanism of development and ways of spreading the disease. In the intestine of man and animals, oncospheres are formed from eggs, which, with blood, penetrate into various organs, including the brain and spinal cord. Further for 2,5-3 months in the organs the invasive stage of the bladder is formed-the priceus. By eating the brain and spinal cord of the affected victim, animals become infected. In them, a mature mutipse, producing eggs, grows.

On the second day after infection with eggs, traces of larvae( "drill holes") are found in the liver, heart, kidneys and other organs. After 48 hours from the onset of infestation in the brain, hyperemia, infiltrates are detected, the volume of the fluid in the brain ventricles increases. On the 15-20th day, bubbles with price points are already found. On the 40-100th day, the bladder increases, presses on the surrounding tissue and skull bones. There is atrophy of the brain and thinning of the skull.

In the last stage of development, an untreated priceus occupies 2/3 of the skull cavity.

Symptoms of the disease. 2-3 weeks after infection, some patients experience excitement, fear, convulsions. Develops edema and pinpoint hemorrhages in the nipples of the optic nerves. These clinical signs may gradually disappear after 3-6 months. Then the typical symptoms of the "twig" develop rapidly, and the disease passes into the final stage of development.

If the price of the locus is localized in the frontal lobe of the brain, frontal headaches develop, the head is tried to lower it forward. When localization in the temporomandibular lobe of the brain, there is a turning of the head toward the affected lobe, with the location in the occipital lobe - tilting the head to the back, cramps of the legs, hands. The location of the prices in the spinal cord is accompanied by soreness in the sacrum, a shaky gait. In humans all these symptoms develop slowly. Human immunity partially copes with perishing larvae. With weakened immunity, the disease develops rapidly and leads to a fatal outcome. Thus, the course of the disease passes in three periods( the first is acute, the second is the latent and last) of the development of characteristic symptoms, when the price reaches a large size.

Diagnostics of the .During life, the diagnosis is made on the basis of communication with animals and the symptoms of the disease, as well as with the help of an allergic reaction( vaccine only in veterinary doctors) and examination of the fundus. In patients with brain damage, stagnation occurs in the nipple of the optic nerve.

Treatment by methods of orthodox medicine. With a price tag, an anthelmintic panacur( from a veterinary drugstore) is prescribed, especially in the early stages of infestation. The dose of the drug is 0.025 g per 1 kg of body weight for three days orally with water in a ratio of 1: 5-1: 10.

In the absence of the effect of drug therapy in the late stage, surgical treatment can be used.

Injection of a 5% iodine solution into the cavity of the price of the bubble with preliminary suction of liquid from it is also applied.

Early diagnosis is the guarantee of a complete cure.

The causative agent of dirofilariasis is dyrofilaria - round worms. These are small, thin tissue worms, also called filaments.

The final hosts of the dyrofilariae are dogs in which adult parasites are in the subcutaneous tissue. Further development occurs in mosquitoes, which transmit the invasion to humans. Dirofilariasis is a widespread infection of dogs all over the world, and cases of human injury are recorded everywhere, including in Russia.

Clinical manifestations and mechanism of disease development. Larvae of dirofilaria in humans cause the formation of nodes under the skin and mucous membranes, they can be localized in any parts of the body, but more often in the chest, hands and feet, as well as under the conjunctiva and in the orbit. The larva of the dirofilaria repens in the period from 1 month to 2 years from the moment of infection turns into an adult individual, which in the human body is not capable of generating microfilariae, but can form a parasitic tumor containing a round worm 1-3 mm in diameter and 3-8 cm long.

Specific symptoms in some patients is the migration of the worm, manifested by the movement of the tumor under the skin( several centimeters per day).Possible suppuration of the tumor and development of an abscess.

Diagnostics. Laboratory diagnosis is not developed. The diagnosis is established when the worm is removed from the node.

Treatment by methods of orthodox medicine - surgical.

Huge importance of the diagnosis of WFD in the study of patients with a diagnosis of the "cancer" sentence.

Hence the following conclusions.

1. Patients with any tumors need to confirm the diagnosis of "cancer" by other methods, require surgeons to first carry out a morphological study of the removed tumor, and then histological. It is important to remove from the patients the stigma of a terrible diagnosis.

2. Recognition of the infectious-parasitic nature of tumors eliminates all inconsistencies in the clinic, diagnoses, diseases of unknown nature, will significantly advance the results of treatment to recovery.

3. Recommended treatment: HRT and surgical if it brings cosmetic inconvenience.

Causes of the disease. The causative agent is the nematode of the genus dyrofilaria imitis.

Parasitizes many mammals, but the main reservoir of infestation is dogs. Adult worms are localized in the right ventricle of the dog's heart, and microfilariae circulate in the peripheral blood. Further development of the dysrophilia imitis occurs in mosquitoes, which transmits invasion to humans.

The person is an accidental host. Larvae that enter the body are localized in small vessels of the lungs;the invasion of dogs is widespread, therefore cases of human defeat are registered everywhere.

Clinical manifestations and mechanism of disease development. Most cases of infestation with imif larvae of the dystrophy are asymptomatic, pathological changes are detected accidentally by X-ray examination. Sometimes there are pains in the chest, cough, less often - hemoptysis. When X-ray examination, there are "coin-like" lesions in the lungs, globularly limited solitary darkening of 1-2 cm in diameter. In such cases, diagnoses are either "tuberculosis" or "cancer."

Treatment is not developed.

Of the 15 people examined with the diagnosis of "pulmonary tuberculosis", only 2 people showed tuberculosis bacteria on the WFD;in 5 cases fungi were detected, in 3 cases - toxocariasis, in 3 cases - ascaridosis and in 2 cases - dystrophilia worms.

From this case, we can draw the following conclusions.

1. In no case can you unconditionally trust the diagnoses established by orthodox medicine.

2. In connection with the large migration of both populations and bloodsucking insects, to conduct diagnostics of the WFD more thoroughly on all microorganisms incorporated in the apparatus program.

3. In complex and controversial cases, conduct differential diagnosis by testing the therapeutic frequencies of various pathogens with electromagnetic inductors on the problem organ. Do this at least 3 times.

4. An accurately diagnosed diagnosis is not only a cure for dirofilariasis, but also an opportunity to avoid severe diagnoses and, as a consequence, the use of toxic drugs.

A lot of scientific literature on echinococcosis has been written, but more and more patients with cancer diagnosis are coming in, who have clinical signs of echinococcal cysts, and echinococcosis is being tested in the WFD.

They tend, as a rule, already at a late stage.

Even in the official sources of educational medical literature it is indicated that in 80-90% of cases the diagnosis of cancer is made erroneously.

Sbiten is a traditional Russian drink, prepared with the addition of a large number of spices and spices. Due to this, it has a powerful antiparasitic effect, because the ginger, cinnamon, cloves, pepper, cardamom, bay leaves, etc., which it contains, simply do not leave the parasites alive.

Simple sbiten

You will need: 1 kg of honey, 20 g of hop, 5-7 g of cinnamon, cloves, lemon balm or mint. Honey is dissolved in 4 liters of boiling water, then add spices and herbs, put on fire and boil for 2-3 hours. Drink hot, in the evening, before dinner.

Sbiten "Suzdal"

You will need: 150 g of honey and 15 g of dried spices - cinnamon, ginger, cardamom, bay leaf.

Honey needs to be dissolved in 1 liter of water, put on fire and boil for 20 minutes, then add spices and boil for another 5 minutes. You can also add stevia herbs to taste( sweet herb, sugar substitute).Then strain sbiten through gauze and drink hot, before dinner.

Sbiten "Vladimirsky"

You will need: 200 g honey and 5 g cloves, cinnamon, ginger, bay leaves. Honey is dissolved in 1 liter of water, boil for 20 minutes, then add the spices and boil for another 5 minutes. Strain through the cheesecloth, in the evening before dinner drink in a hot form.

Such sauces, possessing antiparasitic action, are well suited to any dishes.

Sauce with apple juice

You will need: 1 tbsp.spoon crushed walnuts, 1 tbsp.a vegetable spoon is small( better pumpkin or walnut oil), 3 tbsp.spoons of apple juice.

Peel the nuts until smooth. Pour in the vegetable oil and stir until a thick mixture is obtained. Blend the mixture with the juice of sour apples.

Sauce with nuts

You will need: 100 g of ground nuts, 100 g of vegetable oil, 1 tbsp.spoon of lemon juice, 1 tbsp.a spoon of grated onion, one grated clove of garlic, grated peel of one lemon.

Peel the nuts until smooth. Add onion grated on a small grater, garlic, then pour in the vegetable oil to get a thick mixture.

Season with lemon juice and finely grated lemon zest.

Sauce with pumpkin seeds

You will need: 100 g of purified pumpkin seeds, 100 g of vegetable oil, 1 tbsp.a spoon of tomato puree, 1 tbsp.spoon of lemon juice, 1 tbsp.a spoon of grated onion, finely grated peel of one lemon

Peeled seeds to pound until a homogeneous mass is obtained. Add the tomato puree and grated onion. Stirring the resulting mixture, in small portions, gradually introduce vegetable oil into it, so that the mixture turns thick and lush. Mix the mixture with juice and lemon rind.

Sauce with soy flour

You will need: 2 tbsp.spoons of soy flour, 6 tbsp.spoons of silicon water, 100 g of vegetable oil, 1 tbsp.a spoon of lemon juice, 1 teaspoon of dried brewer's yeast, tarragon.

Soy flour mixed with water. The resulting dough, stirring constantly, combine with vegetable oil. Season with lemon juice, beer yeast and finely chopped tarragon.

Sauce with lemon juice

You will need: 2 tbsp.spoons of vegetable oil( preferably walnut oil), 1 tbsp.spoon of lemon juice, 1 tbsp.a spoon of grated onion, 1 teaspoon of honey.

Beat well the butter, juice and onions. The resulting sauce is suitable for any green salad.

Sauce with lemon juice

You will need: 3 tbsp.spoons of vegetable oil( give preference to oil from pumpkin seed), 1 tbsp.spoon of honey, 1 tbsp.spoon of green coriander.

Mix oil, honey and lemon juice. Add the chopped coriander.

Tomato sauce with nuts

You will need: 1 large ripe tomato, 1 tbsp.a spoon of pounded walnuts, parsley or celery.

Peel well of ripe tomatoes. Grate them on a grater or whip with a mixer. Add nuts and finely chopped parsley or celery.

Nut sauce

You will need: 4 tbsp.spoons of ground nuts, 2 cloves of garlic, 1 tbsp.spoon of lemon juice.

Peel the nuts until a uniform gruel is obtained. Rub garlic and combine with "nuts".Mix the mixture with lemon juice.

Sauce with nuts and bread

You will need: 1 tbsp.a spoonful of walnuts, 1 slice of black bread, 3-4 cloves of garlic.1 tbsp.spoon of vegetable oil, 1 tbsp.spoon of lemon juice, 1 tbsp.spoon of apple juice.

Nuts to pound until a uniform gruel is obtained. Bread soak in water, squeeze well and crumble into nuts. Beat the resulting mixture, add the mashed garlic and, stirring constantly, pour the lemon juice. Add the vegetable oil. Dilute with apple juice. Good shake.

Sauce with ginger and peanuts

You will need: 1 cup of peanuts, 2 tbsp.spoons of ginger, 1 tbsp.spoon of apple juice.

Peel the peanuts until smooth and mix with ginger. Allow this mixture to stand for 10-15 minutes, then whisk thoroughly. Add apple juice( sour) to the mixture.

Sauce with onion

You will need: 1 pc.onions, 2 tbsp.spoons of sour apple juice, 1 glass of peeled walnuts.

Finely chop onion and mix with ground nuts, add apple juice.

This sauce can be spread on bread, cabbage leaves, potatoes.

Sauce with condiments

You will need: 2 tbsp.spoons of grated sour apples, 1 glass of peeled walnuts, 1 tbsp.a spoon of mixed seasonings( parsley, ginger, cloves, cumin, sweet red ground pepper, thyme, celery greens).

In the ground nuts add grated apples and condiments. This sauce is good as a seasoning for fresh vegetables.

Red sauce

You will need: 0,5 kg of tomatoes, 3 tbsp.spoons of olive oil, 2 cloves of garlic, 1 tbsp.a spoonful of tomato paste, a quarter of a tablespoon of ginger, a pinch of salt, sugar.

Peeled tomatoes in a vegetable oil, add finely chopped garlic and tomato paste. Tumble on fire for about 10 minutes. Wipe through a sieve and evaporate. Season with ginger, salt and sugar.

Tomato sauce with mushrooms

You will need: for 1.5 glasses of tomato sauce - 75 g of champignons, 2 onions, 1 tbsp.a spoonful of vegetable oil, a teaspoon of ginger, salt to taste.

Prepare tomato sauce. Chop the mushrooms. Finely chop the onion, lightly fry. Mushrooms and onion pour tomato sauce. Put on the fire and boil for 15 minutes.

Remove from heat, season with salt, ginger.

This sauce is good for fish dishes.

Apple vinegar

You will need: 400 grams of crushed apples taken 0.5 liters of water, 100-200 grams of honey( but you can sugar), 10 grams of yeast, 20 grams of dry black bread.

Apples peeled, cut out the core. Cut into pieces and grind in a mixer or grater. In the apple mass pour boiled water, add honey( sugar), yeast and dry black bread. The mixture should be placed in a vessel with a wide neck, covered with a clean cloth and put in a dark place( room temperature should be at room temperature).

Stir the mixture 2-3 times a day for 10 days. After this period, strain the composition through the cheesecloth. For each liter of liquid add from 50 to 100 g of honey( you can sugar).Close the gauze container and put again in a dark place to continue fermentation. When the liquid brightens, it will be a signal that fermentation is over. The period of readiness is from 40 to 60 days.

Finished vinegar must be filtered, bottled, corked. Vinegar should be stored in a cool place.

Vitamin Vinegar

You will need: 1 l berries, 1 l of cold water, 150 g of sugar.

Two-liter jar filled half-chopped gooseberry, white currant or cherry. The first stage: the berries pour 1 liter of cold water( it is better to take silicon water), add 150 g of sugar. The second stage: cover the throat of the can with a clean white cloth and put it for 90 days near the window. To put it is necessary so that the bank was in heat, but also did not fall under the action of direct sunlight. The mixture should wander well. After three months, strain the liquid and pour it over the bottles. Bottles are sealed and stored in a place where they are laid. This vinegar has a delicate taste, full of vitamins. It goes well into vegetable salads.

Spicy white vinegar

You will need: for 1 liter of white fruit vinegar - 1 tsp chopped fresh parsley, 1/2 tsp mustard seed, 1/2 tsp cinnamon, 1/2 tsp ginger, 1 /2 tsp cinnamon, 1/2 teaspoon sweet paprika, 1/2 tsp nutmeg, 1/2 teaspoon cloves, 1/2 teaspoon salt, 1 bay leaf, 1 crushed clove of garlic.

All these ingredients are covered in white fruit vinegar. The mixture is put on the fire and cooked for 2-3 minutes. The mixture is poured into a glass container and allowed to stand for 3 days. After that, the liquid is filtered and bottled.

Spicy red vinegar

You will need: for 1 liter of red fruit vinegar is taken 1/2 teaspoon of ginger, 1/2 teaspoon of ground lemon peel, 1/2 teaspoon of muscat color, 1/2 tsp cinnamon,1/2 tsp thyme, 1/2 tsp black pepper, 1/2 teaspoon sweet red paprika, ground garlic clove and 2 bay leaves.

All listed ingredients fall asleep in red fruit vinegar. The mixture is put on the fire and cooked for 2-3 minutes. The mixture is poured into a glass container and allowed to stand for 3 days. After that, the liquid is filtered and bottled.

Mint vinegar for sauces and sauces

You will need: for 1 liter of fruit vinegar is taken 20 g finely chopped young shoots or leaves of fresh mint.

Ingredients are mixed, poured into a vessel, covered and held for 20 minutes. Bring to a boil 1 liter of fruit vinegar, add to it a mixture of sugar and mint. The mixture is kept on the fire for 3 minutes, continuously stirring. The cooled liquid is placed in a cold, dark place for a week. After this time, the mixture is filtered and bottled.

| Do not forget that all the vegetables and greens that you are going to use in your salads must be very cleanly washed! Salad of dandelions with nuts

You will need: 2-3 handfuls of finely chopped dandelion leaves, 5-6 chopped walnuts, 1/2 teaspoon of ginger, 2 tbsp.spoons of vegetable oil, honey to taste.

Mix the greens with nuts, add ginger. Refill with vegetable oil and honey. And there is a pleasure.

Mixed salad

You will need: 2 handfuls of finely chopped dandelion leaves, 1 handful of finely chopped green salad, 1 handful of finely chopped sorrel, 5-6 radish, 5-6 walnuts, 1/2 teaspoon of ginger, 1 tbsp.spoon of honey, 2 tbsp.tablespoons vegetable oil.

Mix the greens, sprinkle nuts, add radish, ginger rubbed on a large grater. Season with honey and vegetable oil.

Salad of dandelions and nettles

You will need: 1 handful of finely chopped dandelion leaves, 2 handfuls of nettle, 1 tbsp.a spoon of finely chopped onion, 2 cloves of finely chopped garlic, 5-6 chopped walnuts, 1 tbsp.a spoon of pickled cabbage brine, 1 tbsp.spoon of vegetable oil.

Mix the greens, add onion, garlic, walnuts. Season salad with brine and vegetable oil.

Salad from sorrel

You will need: a bunch of sorrel, 5-6 chopped walnuts, a pinch of ginger, 1 tbsp.spoon of honey.

Finely cut the leaves of sorrel with stems, add crushed nuts, ginger and mix it all with 1 tbsp.spoon of honey.

Salad from plantain

You will need: 2 handfuls of young leaves of plantain, 1 tbsp.spoon of vegetable oil, 1 tbsp.spoon of honey.

Finely chop leaves, season with honey and vegetable oil.

Plantain salad with onion, ginger and nettle

You will need: 3 handfuls of young plantain leaves, 1.5 handfuls finely chopped onions, a handful of finely chopped nettle, 1 tbsp.spoon of vinegar.

Cut the greens, season with vegetable oil, apple cider vinegar. Sprinkle with ground nuts, throw a pinch of ginger.

Carrot salad with black olives

You will need: 200 g carrots, 1 tbsp.spoon of vegetable oil, 1 tbsp.a spoon of crushed walnuts, V4 teaspoon of ginger, lemon juice, 4-5 olives.

Wash carrots, peel, grate. Season with vegetable oil, add lemon juice, ginger. Sprinkle with nuts. The dish should be decorated with olives.

Carrot salad with spices

You will need: 2-3 pcs.carrots, a teaspoon of vegetable oil, 6 walnuts, ginger, cinnamon.

Wash carrots, peel, grate. Pour lemon juice, sprinkle with spices, sprinkle with nuts.

salad sauerkraut

you will need: 600 g of sauerkraut, 100 g of pickles, 100 grams of green onion, 100 g of vegetable oil, V4 teaspoon ginger, rA teaspoon flaxseed, salt, sugar, ground black pepper to taste,1 head of onions.

Cut the cabbage, cut the cucumbers into cubes, chop the green onions, season with salt, sugar, pepper, ginger, vegetable oil, sprinkle with flaxseed.

Apple and pumpkin salad

You will need: apples - 200 g, pumpkin - 200 g, carrots - 10-20 g, ginger - 20 g, 1 tbsp.a spoon of lemon juice, honey to taste.

Peeled apples and pumpkin chop on a large grater, mix, add honey, lemon juice, ginger. Salad is put in a salad bowl and decorated with grated carrots.

paste of wild edible herbs

You will need: nettle, dandelion leaf, clover, coltsfoot, plantain, lady's mantle, knotweed, the leaves and flowers of linden, birch leaves and raspberries, honey, ginger, sunflower oil.

All herbs are washed very well and passed through a meat grinder. Then you can prepare two versions of the paste.

Pasta Sweet

Grass mixed with honey through a meat grinder. Such paste can be stored in the fridge for a month, and if you roll up the lid, it will take longer.

Salted pasta

Mixed with herbs mixed with sunflower oil, add a teaspoon of ginger. In this paste, if desired, you can add onions and garlic. This pasta is very good for sandwiches.

A teaspoon of such pasta taken before meals will provide you with a daily dose of trace elements and vitamins.

Caviar bow

You will need: 300 g onion, 30 g sweet pepper, 80 g carrots, 60 g white bread, 60 g tomato puree, 60 g vegetable oil, 20 ml apple cider vinegar., Teaspoonful of flaxseed,greens, salt.

Onion, sweet pepper finely chopped, carrots rubbed on a grater. Onions and peppers are fried, add carrots, tomato puree, grated bread and stew until cooked. At the end of quenching, caviar is seasoned with salt, vinegar and flaxseed.

Shchi green

You will need: 4 potatoes, 1 carrot, 200 g of young nettle, a bunch of sorrel, a bunch of dill, a bunch of parsley, 1/2 teaspoon of ginger.

Finely chop onion and put into a pot with potatoes. Wash thoroughly and cut into greenery. Nettle finely chopped. Sorrel cut into large pieces. Once the potatoes with onions are ready, add nettle to them.

Grated carrot, sorrel and other herbs in a small grater, put in a saucepan, close the lid and let it brew.

Add the ginger and salt( to taste).

Also in green shchi you can add leaves of meadow clover.

Shi lazy

You will need: 500 g fresh cabbage, 3 onions, 1 carrot, 2 potatoes, 3 tomatoes, garlic head, parsley root, celery root, bunch of dill, parsley bunch, 3 bay leaves, 3 peas of sweet pepper, 1 /4 teaspoons of ginger, salt( to taste).

Cut potatoes in half, the root of parsley and onion - into four parts. Throw in boiling water.

Peel the tomatoes. Cut into large slices and put into a saucepan.

Add bay leaf, sweet pea, ginger. The celery root should be rubbed on a large grater and thrown into cabbage soup. Shchi cook less than 12 minutes.

Grate the carrots on a small grater and put into the prepared dish, along with mashed or very finely chopped garlic.

Beetroot soup

You will need: 3 beets, 1 tbsp.spoon flour, 1 tbsp.a spoon of vegetable oil, a pinch of ginger, salt to taste, greens.

Boil beets, peel and grate on a large grater. Mix 1 tbsp.spoon of vegetable oil from 1 st.with a spoonful of flour, put in a frying pan, warm thoroughly, then add the beetroot and put it out a little.

Drain mixture into boiling water. Boil 1-2 minutes, add ginger. Pour into a plate, sprinkle finely chopped herbs.

Borsch with beans

You will need: 1-3 carrots, parsley root, 200 g of string beans, 1/2 small head of cabbage, 2-3 potatoes, 1 onion, 2-3 tomatoes, 2 beets, 4 teaspoon flaxseed,salt, pepper( to taste).

Carrots clean and cut into cubes. Also chop the root of parsley, beans and cabbage. Throw vegetables in boiling water. Wet the beetroot to grate, fry in vegetable oil with onions. Add potatoes, fried beets with onions and tomatoes to the water( peel the tomatoes and fry them in oil, too).Add spices and flaxseed.

Rassolnik

You will need: 3-4 salted cucumber, potato, 1 carrot, 1 turnip, leek, 3 bay leaves.1/2 teaspoon of ginger, a bunch of dill, a bunch of parsley.

Cut the potatoes into cubes and drop them into a saucepan of boiling water. Carrots and parsley root chopped and put in a saucepan, followed by a turn and a turnip, shredded with straw. Pour the onions finely chopped, dip into a saucepan. Add bay leaves and ginger. Salted cucumber peel, cut into four parts, remove seeds. Cucumbers finely chopped( you can use a grater) and put in rassolnik.

Remove pan from fire, add finely chopped greens and allow to stand under the lid.

Pumpkin soup

You will need: half a kilo of pumpkin, one onion, salt, pepper( to taste).Onion finely chopped and fried in vegetable oil until transparent.

Pump the pumpkin and cut into cubes. Put into a saucepan, add onion, a little water and simmer until ready( 4-5 minutes).Wipe through a sieve and dilute with water. Again put on fire, but do not boil.

Solyanka

You will need: a pound of white cabbage, 3 tbsp.spoons of vegetable oil, 1 onion, a bunch of parsley, 1 tbsp.a spoonful of vinegar, 1 sour apple, sugar to taste.

Cut cabbage, add salt and simmer in vegetable oil and salted water. To the cabbage add the chopped onion, parsley, vinegar and if necessary vegetable oil. Before serving, add a large planed apple. Garnish with greenery.

Cauliflower fried in breadcrumbs

You will need: 1 kg of cauliflower, 100 g of walnut oil, a teaspoon of ginger, breadcrumbs, salt.

Diced and prepared cauliflower boil in salted water. Then fry in oil in a frying pan. When frying cabbage, sprinkle it with breadcrumbs, ginger and salt.

Carrots with spices

You will need: 1 kg of carrots, half a glass of vegetable oil, 2 tbsp.spoons of spices( taken in equal parts: savory, marjoram, ginger, chervil, basil), 1 tbsp.a spoonful of sugar.

Carrots to clean, cut in half( in the longitudinal direction), lightly boil. Take a special form for the oven, grease it with vegetable oil, sprinkle the bottom of 1 tbsp.spoon spices( shake the pan so that the spice is distributed evenly).

Put the carrots into a shape, cut them down. Top with the remaining mixture of spices. Sprinkle with vegetable oil and sprinkle with sugar.

Pour 1-2 cups of water into the mold and cook in an oven at 170 ° C until the carrots are ready.

Lentil

You will need: 1 glass of lentils, 1 onion, 5-6 pcs.dried apricots, 5-6 walnuts, 1 tbsp.a spoon of vegetable oil, salt, pepper, ginger, parsley greens.

Lentils cook almost until they are ready, salt, add finely chopped fried onions, nuts, dried apricots, ginger and cook for another 10-15 minutes. After that, remove from heat, sprinkle with pepper and herbs.

Tomatoes with rice

You will need: 10 tomatoes, a glass of rice, 1/2 cup of peeled walnuts, a bulb, a bunch of vegetable greens, 1/2 teaspoon of flax seed, a tablespoon of sunflower oil.

Prepare tomatoes for stuffing. To do this, they need to cut off the top and remove the flesh. Boil the rice. Shred the onion, fry it in vegetable oil. Mix rice, onion, walnuts, flax seed. With this mixture stuff the tomatoes. Put the tomatoes in a deep frying pan, sprinkle with sunflower oil and put in the oven.

Remove from the oven, sprinkle with herbs.

Cutlets from lentils

You will need: 300 g of lentils, 1/2 cup of wheat flour, 10 g of biscuits, 3 cloves of garlic, vegetable oil, U.ch. spoons of flaxseed.

Lentils soaked for 10-12 hours, cook until ready, add flour, oil, flax seed, salt. The mixture is passed through a meat grinder. Add the garlic.

Molded cutlets, breaded in breadcrumbs, fried.

Stuffed zucchini

You will need: zucchini, onions, carrots, greens, garlic, boiled rice, 1/2 teaspoon of ginger.

Zucchini cut into rings, remove the seeds and lay on a baking sheet, fill with a mixture of boiled rice, fried onions, carrots, greens, garlic, ginger. From above, each ring with minced be sprinkled with vegetable oil or sprinkled with breadcrumbs.

Wormwood citrus belongs to the family of Compositae. This is a long-standing steppe shrub 30-60 cm high, with numerous stems, yellowish-brown color. Blossoms wormwood in July-August.

Attention: the plant is poisonous!

Leaves of wormwood are small, greyish-green in color. Flower baskets are numerous, miniature, shaped oblong-ovoid, baskets pointed, greenish-brown in color and similar to seeds. Wormwood has a peculiar smell and bitter taste.

Wormwood citrate grows only in Central Asia.

In medicine, we use grass during flowering, and flower baskets - just before flowering.

Useful properties of wormwood:

Flower baskets contain poisonous lactone santonin( 4-7%), bitter and coloring substances, betaine, choline, essential oil( 2-3%), acids( malic and acetic).

Wormwood has a strong antiseptic, anti-inflammatory, analgesic and bactericidal action.

Apply as an antihelminthic.

Tansy common( wild ash) belongs to the family of Compositae. This perennial herbaceous rhizome plant is 60-120 cm high, with erect stubby, branched stalk.

Blossoms tansy in June-August. Flower baskets are rounded, yellow, composed of tubular flowers, collected in a flat corymbose inflorescence. Leaves on top of a dark green color, from below greyish-green.

Warning: the plant is poisonous!

Leaves alternate, with oblong-lanceolate serrated lobes. The plant has a strong odor.

Tansy ordinary grows in gardens, in sparse mixed forests along river banks, in meadows and fields, along roads almost everywhere.

In medicine, a plant is used during flowering.

Useful properties of tansy:

Tansy flowers contain organic acids( tanacet, gallus, etc.), bitter tanacetin, tannic substances, tar, sugars, gum, fatty and essential oils.

Powder prepared from flower baskets, stimulates appetite, strengthens the secretion of the glands of the gastrointestinal tract and tones up its muscles, improves digestion, increases the separation of

bile and sweat, raises blood pressure. The herb has antipyretic, antispasmodic, anti-inflammatory, analgesic, antimicrobial, wound-healing, antihelminthic effect.

Internal application of tansy ordinary requires great care. Infusion of tansy is contraindicated in pregnancy.

Ginger officinalis is a plant belonging to the genus ginger. This is a perennial herb with a complex root system. Blossoming stems( about 30 cm high) carry a spicate inflorescence on the top, in the sinuses of which flowers sit. From a strongly branched horizontal rhizome grows a few above-ground stems. Depending on the variety, the color of the ginger flowers can be different - from light purple to almost black. The birthplace of ginger is India. In the wild, ginger does not occur. Distributed mainly in tropical countries of Asia.

For medicinal purposes, the rhizome of the plant is used after the growing season. The roots of the plant are excavated, washed, cleaned of bark and dried in the sun or in dryers at a temperature of + 50 ° C.

Useful properties of ginger:

The ginger root contains resins, starch, fatty oils( 3U ° / o), essential oil( 1.5-3%), giving the rhizome a characteristic ginger smell. The burning taste of the rhizome is due to the presence of gingerol resinous substance.

Ginger is a diaphoretic, expectorant, carminative, antiemetic, analgesic, antifungal and anti-trichomonidal, antiparasitic.

Ginger is used for food as a spice.

Coriander seeded belongs to the umbrella family. This is an annual grassy essential oil plant with a height of 30-45 cm. The flowers of coriander are small, white in color. The leaves are regular, with a characteristic odor. Blossoms coriander in June-July;Inflorescence is a complex umbrella with 3-5 rays. Fruits coriander in August-September. Fruit - seed, globose, brown, odorous.

Homeland of coriander Southern Europe, Asia Minor. As a wild plant this plant is found in the south of the European part of Russia and in Central Asia.

In medicine, plant seeds are actively used.

Useful properties of wormwood of coriander:

Fruits of Coriandrum sativum L. contain essential oil( up to 1.2%), fatty oil, a small amount of alkaloids.

Fruits increase the secretion of the glands of the digestive tract;have a choleretic, laxative, analgesic and antiseptic effect;stimulate the regeneration of damaged tissues.

Coriander is used in food as a seasoning.

Attention: the use of preparations of coriander seeded requires great care: overdose can cause a stop of breathing.

Plantain large( plantain common) - a plant belonging to the family of plantain. It is a perennial herbaceous plant with a height of 5-70 cm. Plantain blooms in June-September. The seeds of the plant are small, oval, dark brown in color.

Rhizome short. Fruit is an ovoid multisimple boll. The flowers are small, unattractive, brownish in color. They are collected in dense spicate inflorescences 4-40 cm long. The leaves are ovate or elliptical. Plantain large grows almost everywhere, mainly along roads, in gardens, in vegetable gardens, fields. For medical purposes, grass is most often used during flowering. A seed - immediately after their maturation. The juice of a fresh flowering plant is squeezed and canned.

Useful properties of plantain:

The plantain seeds contain mucus( 8-44%), fatty oil( 15-25%).The content of polysaccharides in flowers is up to 25%, in leaves - up to 10%( decreases with age of the plant), in stems - up to 3%.The leaves contain carotene, acids( ascorbic and citric), vitamin K, mucus( up to 11%), bitter and tannic substances.

Plantain preparations are a good remedy for diseases of the respiratory tract, whooping cough, pulmonary tuberculosis and chronic nephritis. Extract from the leaves has a soothing, hypnotic effect, and also lowers blood pressure. Pharmacological studies have shown that the action of the plantain increases the hemoglobin content in the blood, and also improves the condition of patients suffering from neurasthenia and atherosclerosis. Psyllium can be used in the treatment of dysentery, stomach ulcers. Plantain preparations possess antiseptic, anti-inflammatory, wound-healing, blood-purifying, expectorant, laxative and analgesic action.

Flax seed( common flax) is a plant belonging to the family of flax. This is an annual herbaceous plant 30-60 cm high. Fruit-globular capsule. The seeds are flattened, ovate, brownish-yellow, with a smooth shiny surface. Leaves are regular. The flowers are soft blue, with 5 petals, collected in umbrellas. The plant blooms in June-July.

Flax sowing as a cultivated plant is widely cultivated in the northern and central regions of the European part of Russia and in Siberia.

For medicinal purposes seeds are used that ripen in July-August.

Useful properties of flax seed:

Seeds contain fatty oil, protein substances( up to 30%), carbohydrates( up to 25%), mucus( up to 12%), vitamin A, organic acids, enzymes.

In medicine, a flax seed is used that has a slight laxative, enveloping, emollient, anti-inflammatory and analgesic effect.

Dandelion officinalis( dandelion ordinary) - a plant belonging to the family of Compositae. It is a perennial herbaceous plant 10-30 cm high, with a leafless flower stalk( arrow).Fruit is a seed with a tuft. Flower baskets of golden yellow color. Blossoms in April-August.

Dandelion medicinal grows almost everywhere except the Arctic.

For medicinal purposes, grass and juice are harvested during flowering;and the roots are harvested either in early spring or late autumn.

Useful properties of dandelion:

Roots and leaves contain resin, rubber, polysaccharide, inulin, sugar, fatty oil, mucus, organic acids, calcium and potassium salts, colorants-various carotenoids, vitamins, etc. The roots also contain triterpenoid compounds. In flower baskets and leaves contain carotenoids, triterpene alcohols. The leaves, among other things, contain saponins, vitamin C, iron, calcium and phosphorus.

Roots and leaves excite appetite, improve digestion, increase lactation in lactating women, serve as a mild laxative, expectorant and tonic. All parts of the plant have antipyretic, diaphoretic, choleretic, anthelminthic action. Dandelion has a calming and easy sleeping pills.

Rowan ordinary belongs to the family of Rosaceae. This is a small tree 4-15 m high, with odd-numbered serrate leaves. Flowers are small, white, fragrant. The fruits of mountain ash are bright orange false spines, called berries( although they are not berries), they are collected in a brush. Blossom ashberry in May - early June.

Ashberry grows in the undergrowth of coniferous and mixed forests, along forest edges in the forest and forest-steppe zones of the European part of Russia and in the Caucasus. It is often cultivated as an ornamental plant.

For medicinal purposes, fruits of mountain ash are used, which are harvested in September-October.

Useful properties of mountain ash:

Seeds of mountain ash contain fatty oil and glycoside amygdalin. Fruits - sugar, acids( citric, malic, wine and ascorbic), alcohol sorbitol, cyanine chloride, tannic and bitter substances, carotenoids, carotene( up to 18 mg%), vitamins C and P, essential oil. In leaves of mountain ash there is a lot of vitamin C( up to 200 mg%).

Fruits have antiscorbutic, mild laxative, diuretic, astringent, anti-inflammatory and hemostatic effect.

Garlic belongs to the family of lily. This is a perennial bulbous plant. The leaves of garlic are narrow, linear, up to 1 m long. Flowers are small, grayish-white, collected in an umbrella.

Garlic blossoms in July-August. Garlic is widely cultivated as a vegetable plant throughout Russia. Without it, there is not one garden.

Useful properties of garlic:

For medical purposes, bulbs of a plant are usually used. Garlic contains an essential oil( 0.2-0.3%), which determines the specific garlic odor, and this essential oil has a strong bactericidal effect. Even in garlic there are fatty oils, inulin, ascorbic acid, vitamins D and group B, etc.

The use of garlic increases the resistance of the body to colds and infectious diseases. Also, garlic is taken as a means to reduce fatigue. It stimulates appetite, improves digestion, suppresses the processes of putrefaction and fermentation in the intestines, has an analgesic and soothing effect on the intestines.

Onion belongs to the family of lily. It is a two-year herbaceous plant with flattened-globular bulbs. Fruiting in August-September.

The leaves are lancet, hollow, they are usually called feathers. Flowers are small, composed of 6 white petals. Fruit - a box containing black trihedral seeds. Onion is cultivated everywhere.

For therapeutic purposes use bulbs.

Useful properties of onion:

Bulbs contain sugar( 14% - fructose, sucrose), inulin, carotene, vitamins C, B, E and PP, mineral salts( potassium, phosphorus, iron, etc.).Bulbs and leaves contain essential oil, as well as containing compounds, iodine, organic acids( malic and citric).

Onions stimulate appetite, stimulate the functional activity of the gastrointestinal tract, is an antiscorbutic agent.

It should be remembered that abuse of onions can cause complications in diseases of the kidneys, liver, heart, stomach.

Walnut walnut belongs to the walnut family. This monoecious tree is up to 25 m high. The fruit is a drupe, green. It is large, with a wrinkled bone. These stones and call a walnut. The leaves of the tree are complex, odd-pinned. The walnut blooms in April-May.

Walnut grows in mountain areas, in mixed broad-leaved forests of Central Asia, Ukraine and the Caucasus. It is also grown in gardens.

For medicinal purposes, mainly used leaves( collected during flowering), green pericarp( collected in August);fruits( collected in September, after their maturation).

Useful properties of walnut:

Green outer pericaroon of immature nuts contains tannins( up to 25%), coloring agent yuglon, which has a bactericidal action, and vitamin C( 1000-3000 mg%).The leaves contain hydrochloride, tannins, mineral salts, vitamin C, carotene, essential oil( up to 0.03%).

Leaves have antiseptic, tonic, restorative, astringent, mild laxative and antihelminthic effect;improve metabolism in skin diseases, resolve infiltrates, suppress inflammatory processes.

Grapefruit is a family of rut. It is a fruit tree up to 12 m high, with a grayish-brown smooth bark and small thorns. Leaves are regular, oval, shiny, with winged petioles. Fruits grapefruit in December. Pulp of fruit yellow or greenish-yellow, juicy, fragrant, bitterish-sweet to the taste. The flowers are white, fragrant, solitary or collected in a brush. Blossoms the grapefruit in May.

Grapefruit is bred in subtropical areas, including on the Black Sea coast of Georgia.

As a medicinal raw material, fruits and fruit juice are mainly used.

Useful properties of grapefruit:

Fruits retain their vitamin value for a long time. Fruits contain sugars( 3.86-6.78%), organic acids( 1.42-2.38%), mineral salts, pectin substances, vitamins A, B, D, P and C.

Grapefruit has high dietary and curative qualities.