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Primary and secondary kidney nephrosclerosis: the difference between them

  • Primary and secondary kidney nephrosclerosis: the difference between them

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    Renal nephrosclerosis is a disease in which the proliferation of connective tissue is observed in the kidneys. This leads to compaction of the kidneys, and further to their deformation or wrinkling. Isolate primary and secondary nephrosclerosis.

    Primary nephrosclerosis

    This type of disease is the result of impaired circulation in the kidneys, which is caused by a narrowing of the lumen of the kidney vessels. Lack of blood leads to hypertonic or atherosclerotic nephrosclerosis.

    Hypertonic nephrosclerosis can be benign or malignant. For the first, atrophic changes in nephrons and the involvement of the connective tissue stroma are characteristic. In the second case, fibrinoid necrosis of the capillary glands of the glomeruli and arterioles, as well as the protein degeneration of the tubular epithelium, develops. This type of disease quickly leads to kidney failure, which in the absence of hemodialysis leads to a fatal outcome.

    The cause of atherosclerotic nephrosclerosis is the narrowing of the renal artery of an atherosclerotic plaque, resulting in the kidney becoming coarse-grained. In this case, the kidney can continue to function normally, since often the parenchyma remains intact.

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    Secondary nephrosclerosis

    The secondary form of nephrosclerosis is the result of dystrophic and inflammatory changes that have occurred due to the course of diseases such as pyelonephritis, glomerulonephritis, tuberculosis, diabetes, syphilis and several others.

    The main symptoms of kidney nephrosclerosis:

    1. cardiovascular disorders( significant increase in blood pressure, left ventricular hypertrophy of the heart, attacks of angina pectoris, cyanosis of the extremities, gallop rhythm);
    2. changes in the fundus, which manifest themselves as angiospasm, edema of the papilla of the optic nerve, degenerative-infiltrative changes in the retina;
    3. progressive anemia;
    4. can have such signs of kidney nephrosclerosis as general exhaustion of the body, the acquisition of a pale yellow skin by the skin of the face;
    5. urine analysis shows a decrease in its specific gravity, albuminuria, hematuria( their level depends on the course of the disease).

    Features of treatment of renal nephrosclerosis

    If the patient was diagnosed with kidney nephrosclerosis, then immediately begin treatment of the disease. For this purpose, usually prescribed drugs that have anabolic, hypotensive and diuretic effect. Treatment of renal nephrosclerosis includes the reception of enterosorbents, which allows you to remove accumulated nitrogen from blood and tissues, as well as ganglion blocking agents. When taking antihypertensive drugs should carefully monitor blood pressure, so as not to provoke a sharp drop.

    If malignant nephrosclerosis is diagnosed, embolization of the renal arteries is recommended. Depending on the degree of organ damage, either the entire kidney or only a part of it can be removed. After the operation, hemodialysis is mandatory. It is also possible to transplant the donor kidney.

    Diet rules for nephrosclerosis

    Compliance with the diet for renal nephrosclerosis is one of the components of successful treatment of the disease. Particularly relevant is the diet, if the patient has an increase in the level of creatinine and urea, because it indicates the occurrence of kidney failure.

    To relieve the load from the kidneys, it is recommended to almost completely stop using salt, as well as products containing protein. Include in the diet can only products such as low-fat meat and poultry, fish, pasta, cereals, potatoes.

    It is strongly recommended not to eat cheese and cottage cheese, as well as foods high in potassium( bananas, dates, dried apricots, apricots, grapes, prunes).



    Stabilize the condition with walnuts, pine nuts, buckwheat, sea kale, mineral water "Essentuki- 4 ", because they contain a much needed element, like magnesium.

    Restrictions on the amount of fluid being drunk are superimposed depending on the stage of the disease. In the absence of concomitant cardiovascular diseases, it is allowed to consume up to three liters of liquid per day( soups, liquid porridge, vegetables, fruits are also taken into account).If the condition worsens, it is desirable to limit the amount of liquid to 1.5-2 liters per day.

    As a supporting agent, a decoction of cranberries was a great success. To do this, 2 tablespoons of crushed red bilberry leaves are poured with 2 glasses of water. The broth should be boiled for about 15 minutes on low heat and take half an hour before meals to 100 grams.

    Daily intake should be divided into 5-6 meals.

    As practice shows, even with correctly and promptly conducted treatment, as well as the observance of a diet to return the kidney the previous size is already impossible. Therefore, all medical and preventive measures aim to only stabilize the condition and prevent the progression of the disease.

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