Causes of cardiac muscle atrophy, signs and treatment

Atrophy of the heart muscle is a pathological process that does not have an inflammatory nature, as a result of which a change in the structure of myocardial cells occurs. The second name for this disease is myocardial dystrophy. It arises because of the violation of metabolic processes and leads to a weakening of contractility, as well as other functions of the myocardium and the development of heart failure.

This pathology is most common in the elderly, as the processes in the body at this time begin to slow down, there is a gradual involution in all organs and systems. In this case, this process affects not only the muscle of the heart. But muscle atrophy in the heart can also arise as a result of certain diseases.

Causes of Atrophic Changes in the Heart

All the reasons for the development of cardiac muscle dystrophy can be divided into two categories - congenital and acquired. A direct change in the cellular structure of the heart cells is observed in congenital cardiomyopathies, the origin of which has not been sufficiently studied.

There is also a number of pathological processes underlying the development of myocardial dystrophy that occur in the process after birth. These include:

• Intoxication. Occurs as a result of acute or chronic poisoning of the body with tobacco, alcohol, drugs, drugs, inferior foods, industrial substances and other toxic compounds that enter the body.
• Infection. Often the atrophy of the heart muscle arises against the background of acute viral( influenza, Coxsackie virus) or chronic infection. Especially often complications on the heart give constant inflammatory processes in the nasopharynx.
• IHD.It arises as a result of chronic myocardial ischemia against the background of pronounced coronary atherosclerosis.
• Excessive overload( in athletes and people in manual work).It appears as a result of a constantly increased strain on the heart, with which it can not cope. This is expressed in the violation of oxygen metabolism, including in cardiomyocytes.
• Deviations in the work of endocrine organs( thyrotoxicosis, hypothyroidism).
• Chronic anemia, beriberi, starvation.
• Metabolic disorders and obesity.
• Hypodinamy.
• Pathological processes in the liver and kidneys.
• Psychosomatic abnormalities.
• Dysfunction of the digestive system( cirrhosis, hepatitis, pancreatitis).

How myocardial atrophy manifests itself and its diagnosis

With the development of cardiac muscle atrophy, especially in the initial stages, the symptoms of the disease may be absent. But most often patients complain of pain in the chest on the left, the occurrence of dyspnoea with exercise. There is a feeling of palpitations or irregularities, pronounced weakness and decreased ability to work.

Symptoms of cardiac muscle atrophy are also manifested in the form of edema, which along with shortness of breath are a fairly reliable sign of the development of heart failure. The weakening of the suction function of the heart leads to stagnant phenomena in the veins of the lower extremities, the sweating of the plasma through the venous wall and results in the phenomenon of puffiness, which intensifies toward evening and decreases in the mornings.

Disturbances in the lungs are manifested in the form of increased dyspnea and the appearance of a cough with profuse phlegm at night. This is due to the inflow of blood from the lower limbs when the body moves to a horizontal position, and the increased stress on the heart. During a cough in sputum, traces of blood can be detected, this is due to overflow and rupture of bronchial veins.

In advanced cases, discomfort is added to the right upper quadrant, this is due to stagnation of blood in the liver and stretching of the capsule of this organ, and then ascites( fluid in the abdominal cavity).

Rhythm disturbances, which are accompanied by dystrophic processes in the myocardium, arise due to the death of cells of the conduction system of the heart. In this case, there are many ectopic foci of excitement, causing bradycardia, and various arrhythmias.

With the progression of dystrophic changes, all the symptoms intensify, puffiness does not pass even at night, and dyspnoea is already disturbed at rest.

To clarify the diagnosis, a general examination is conducted to identify the cause that caused the disease atrophy of the heart muscles. To do this, a complete set of standard studies is done - general blood and urine tests that will help to identify the presence of a chronic infection, to assess the level of hemoglobin. When suspected of atherosclerotic changes in blood vessels, a lipidogram is prescribed.

Evaluation of the conductive system and the presence of ischemic changes in the myocardium allows ECG and Holter monitoring. On echocardiography with myocardial dystrophy, the expansion of the cavity of the organ and the reduction of the contractile function are determined. Such a study allows you to see and cicatrical changes in their presence.

If the causes of the development of cardiac muscle atrophy are associated with endocrine pathology or other diseases, the patient is prescribed a consultation of narrow specialists to clarify the diagnosis and supplement the necessary medications and recommendations to a set of therapeutic measures.

Methods of therapy and prevention of muscle atrophy of the heart muscle

Treatment of the disease in old age consists in the application of symptomatic therapy and maintenance of myocardial function.

When identifying the underlying disease, all efforts should be directed to its elimination or transfer to the stage of remission, the mandatory step is the sanation of foci of chronic infection.

Atrophic processes in the cardiac muscle tend to be reversible only at the initial stages of the process. Therefore, timely treatment for help and adequate treatment can lead to complete recovery.

Of the drugs designed to improve the metabolism of myocardial cells, polyvitamins, ATP, trimetazidine, and mildronate are often prescribed.

Effective treatment of myocardial muscle atrophy is possible only in the initial stage of the process. In the future, any therapy performs only a supporting function. The patient must comply with the regime of the day, the restriction of the load, adequate nutrition with the restriction of salt with pronounced swelling.

Of drugs for the development of heart failure, in any case, ACE inhibitors are prescribed( to protect target orans and improve the clinical state of the patient), with severe edema, diuretics are used. In small doses, the appointment of cardiac glycosides is indicated, and with the development of atrial fibrillation they are the drugs of choice. Sometimes it is recommended to use beta-blockers.

Attachment of angina pectoris requires the use of peripheral vasodilators( nitrates), severe rhythm disturbances are stopped by antiarrhythmics, with the transfer of a heart attack, it is mandatory to take aspirin or indirect coagulants to prevent thrombosis of coronary vessels. Statins are often used to control cholesterol levels, as well as omega-3 polyunsaturated fatty acids.

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