Treatment of cholesterol with folk remedies

According to the "aquatic" theory, increase in the level of cholesterol is a sign of the inclusion of the protective mechanism of cells against the drawing of water from their membranes. Simply put, this is the state of the body, in which too much dense blood can no longer extract water from the cells, since in this case their functions are violated. Cholesterol in this case plays the role of a mortar, which, hitting the intercellular space, does not allow water to pass through the cell walls.

Thus, the excess production of cholesterol and its deposition in cell membranes is part of the program to protect cells from dehydration. In retained functions and nucleated cells, cholesterol regulates the flow of water through their membranes. In cells that perform their functions, but do not have a nucleus, the fatty acids that make up the membranes create the conditions under which the cell is able to survive dehydration. Thus, the production of cholesterol by the membranes of cells is a way of their survival under unfavorable conditions. Therefore, cholesterol is a vital substance for the body, and its excess in the blood testifies only to dehydration.

It is water that gives shape to "bricks", of which cell membranes are composed. She's holding them together. If the membrane is dehydrated, the water stops performing its functions and begins to seep through the holes in the membrane. And now imagine that you are sitting at dinner and you are served a dish. If before the meal you do not drink water, the process of digestion will negatively affect the cells of the body. After all, in order to break down proteins into amino acids, water is simply necessary. The intestine also needs water, in order to process food components and deliver them to the liver.

There, special cells are also included in the digestive process, after which the nutrient-enriched blood is sent to the heart. Liver cells also need a lot of water to process the food ingredients that have entered it.

Enriched blood from the liver to the right side of the heart is first pumped to the lungs to enrich with oxygen. Simultaneously, it is dehydrated, since some of the water evaporates at the same time.

Then, concentrated blood is pumped to the left side of the heart, and then enters the arterial system. First of all, oxygen and nutrients are needed for large blood vessels and capillaries of the brain and heart. In places where the arteries are bent, cells undergo strong blood pressure, and they need to protect themselves from damage.

Finally, the time comes when the brain, evaluating the body's water resources, realizes that there is not enough water for everything, and makes its "master" drink water. However, in the body, she acted too late: the damage caused to him already felt the cells of the blood vessels. But if we understand that water is the most important component of the diet, and we drink it before eating, we will get a powerful weapon against the cholesterol deposits in our vessels.

After a long daily intake of water, the cells will suffice moisture, and the need for cholesterol protection will disappear by itself. After that, the cholesterol level will decrease. In addition, fat-burning enzymes, very sensitive to various kinds of hormones, after a walk for an hour begin to intensify and act for 12 hours. Thanks to such walks and lowering of cholesterol level in the blood, the process of fat burning starts. As a result, cholesterol plaques are destroyed and arterial passages are released from this obstruction. Walking is desirable to be done twice a day, thus supporting the activity of lipase( the fat-burning enzyme).

Thus, an increase in the level of cholesterol in the blood and blockage of blood vessels with cholesterol plaques is nothing but a consequence of dehydration of the body. It is the lack of water, not the excess cholesterol that can cause many degenerative diseases. Therefore, it will be more reasonable to take care of regular consumption of water than about diet food. If the enzymes show normal activity, our body is able to cope with almost any food, including that containing cholesterol.

If, when treating water, the cholesterol level first falls, and then starts to rise again, make sure that your body has enough salt.

Cholesterol in the body serves as a building material for many types of hormones. Therefore, the stimulation of hormone production can also increase the production of cholesterol. In addition, at a certain age, chemicals similar to cholesterol, hitting the bloodstream, passing through the lungs, can narrow the lumen of the heart arteries. This leads to pain in the heart, known as anginal pain. The same substances can contribute to the narrowing of the vessels and the deposition of cholesterol plaques on their walls. Nevertheless, the main cause of conditions classified as lung and heart disease is dehydration.

In scientific circles, the prevailing view is that a high level of cholesterol in the blood directly contributes to the clogging of the arteries and thus leads to myocardial infarction and stroke. Among residents of those countries where the average content of cholesterol in the blood is small, coronary heart disease is rare. But in one of the regions of Finland, where there is the highest level of cholesterol, the frequency of myocardial infarction is the largest in the world. Ask your doctor what your cholesterol level is, and if it is higher than 5.2 mmol / l, do everything to make it lower. Ideally, the cholesterol content in the blood should be in the range of 4 to 4.5 mmol / l.

Where does cholesterol in the blood come from?

Our blood supplies cholesterol food of two kinds: already containing cholesterol and that containing "saturated" fats, it is basically animal fats. Four groups of food products contribute to the formation of high levels of cholesterol in the blood:

- the cholesterol content is very high in egg yolks and by-products( liver, kidneys);

- a lot of saturated fat and a lot of cholesterol in beef, lamb, pork and meat semi-finished products;

- saturated fats - in all dairy products, except skim, in particular, a lot of milk fat in cream and sour cream. Coconut, palm oil and products containing vegetable oils that have undergone curing or hydrogenation contain a lot of saturated fats.

What should I do?

Actually, do not set as your goal to change the food too quickly. After all, if unhealthy habits develop over the course of a lifetime, then you can wait a year or two to gradually wean them. Set the task - gradually abandon the food of animal origin and accustom yourself to the plant. Maybe you'll have to look for new recipes, but as a result, you will save not only money, but also health. What, specifically, do you need to do?

Limit the intake of egg yolks to three per week.

Do not eat liver or other by-products more than once a month. Eat less products such as smoked sausages, sausages, or discard them altogether, instead of red meat several times in a week, prepare a vegetable-rich vegetable dishes, fish, chicken or turkey, choose cheeses made from non-target milk, and cottage cheesewith reduced fat, less drink whole milk, cream and sour cream,( switch first to low-fat milk, and when you get used to its taste, skip skim milk), try not to eat butter( go to somedietary margarine).With regard to margarines, it is better to those on which the first component indicates liquid vegetable oil.

Avoid saturated fats:

lard( lard), butter, milk fat, meat fat, meat fat, hydrogenated vegetable oils, hardened vegetable oils;unsaturated fats - good in moderation, safflower oil, cottonseed oil, soybean oil, corn oil, sunflower oil, sesame oil, peanut oil, olive oil.

Cholesterol-lowering products in the blood

Recently, scientists have determined that certain substances contained in natural foods can really help reduce cholesterol.

One of these substances is "guar gum", which is in beans and oats. Another is pectin in apples and citrus pulp. Some researchers argue that other substances from natural foods also have such properties. But there is no evidence that a protective effect can be obtained by introducing lecithin, garlic or vitamins into the diet, just as it is not proved that lecithin, which is found in eggs, protects against cholesterol in the same eggs and in large quantities.

Total cholesterol in serum

HS is a secondary monohydric cyclic alcohol. Chronicles enter the body with food, but most of it is formed endogenously( synthesized in the liver).ChS is a component of cell membranes, a precursor of steroid hormones and bile acids. At least 10% of the population suffer from hypercholesterolemia. By itself, hypercholesterolemia is asymptomatic, but can lead to serious pathological changes in the walls of blood vessels in vital organs.

Concentrations in the blood of cholesterol and TG are the most important indicators of lipid metabolism in patients. They provide important information for further tactics of diagnosing lipid metabolism disorders, solving the issue of hospitalization, choosing a method of treatment and evaluating its effectiveness. The reference values ​​of the concentration of total cholesterol in the blood are presented in Table.[Tiz U., 1997].The concentration of cholesterol above 6.5 mmol / l is considered a risk factor for the development of atherosclerosis. There is a relationship between the increase in the concentration of cholesterol in the blood and the risk of developing CHD.In people at risk of coronary heart disease, the definition of Cholesterol in the blood is recommended to be performed every 3 months.

• The content of cholesterol in the blood increases with type I, IIA, 11B, III, IV, V, polygenic hypercholesterolemia, familial combined hyperlipidemia, primary exogenous hypertriglyceridemia, liver diseases, intra- and extrahepatic cholestasis, pancreatic and prostate cancers, glomerulonephritis,hypothyroidism, nephrotic syndrome, chronic renal failure, alcoholism, isolated somatotropic hormone deficiency( STH), hypertension, coronary heart disease, diabetes, gout, glycogeneses I, III and VI types, large thalassemia, analbuminAI disglobulinemii, Mr. Vernet's syndrome, idiopathic hypercalcemia, OPP.
• Reduction in the content of cholesterol in the blood is noted with a deficiency of a-lipoproteins, hypoproteinemia and abetalipoproteinemia, liver cirrhosis, malignant liver tumors, hyperthyroidism, malabsorption syndrome, malnutrition, sideroblastic anemia, thalassemia, chronic obstructive pulmonary diseases, rheumatoid arthritis, lymphangiectasia of the intestine, megaloblastic anemia. Rapid reduction in the concentration of cholesterol in liver diseases is a poor prognostic sign, often observed in subacute liver dystrophy.
• When evaluating the results of the study of general cholesterol, it should be taken into account that some drugs have a pronounced effect on its concentration in the blood.
• Increase the concentration of cholesterol in the blood: androgens, chlorpropamide, HA, ACTH, epinephrine( adrenaline), sulfonamides, meprobamate, phenothiazines, thiazide diuretics.
• Reduces the concentration of cholesterol in the blood colchicine, haloperidol, monoamine oxidase inhibitors.

When assessing the risk of developing atherosclerosis and its clinical manifestations, the indicators reflecting the content of not only total cholesterol but also cholesterol fractions are often used.

High-density lipoprotein cholesterol in the blood serum

High-density lipoprotein( HDL-XC) cholesterol is defined as the remaining amount of cholesterol in the blood serum after the deposition of apo-B-containing lipoproteins( low and very low density lipoproteins).Blood lipoproteins transport lipids, including cholesterol, from one cell population to another, where they are stored or meta-bolziruyutsya. Unlike other LPs, HDL cholesterol transport of cholesterol from peripheral organs to the liver, where cholesterol is converted to bile acids and is excreted from the body. This is typical for the heart muscle with its vessels, and for other organs.

A decrease in the concentration of HDL cholesterol less than 0.9 mmol / l is associated with an increased risk of atherosclerosis. Epidemiological studies have shown an inverse relationship between the concentrations of HDL-C and the prevalence of IHD.The definition of HDL-cholesterol helps identify the risk of developing coronary artery disease. Reducing the concentration of HDL-Cs for every 5 mg / dL, or 0.13 mmol / L below the average leads to an increased risk of developing CHD by 25%.

Elevated HDL-C concentration is regarded as an anti-atherogenic factor.

Currently, the concentration of HDL cholesterol in the blood serum below 0.91 mmol / l is considered as an indicator of a high risk of coronary heart disease, while a level above 1.56 mmol / l plays a protective role. To determine the tactics of treatment, it is important to simultaneously assess the concentration in the blood serum of total cholesterol and HDL-C.If the patient's HDL-C-cholesterol concentration is lower( less than 0.91 mmol / L) and the total cholesterol is normal, exercise, smoking cessation and weight loss are most effective in preventing IHD.With an increase in the concentration of total cholesterol and a decrease in the level of HDL-C( less than 0.91 mmol / L), medical intervention programs should be aimed at reducing the level of total cholesterol using special diets or, if necessary, with drug therapy.

After determining the content in the blood of HDL-cholesterol, it is possible to calculate the cholesteric coefficient of atherogenicity( Cxc): Kxc =( total cholesterol-HDL-cholesterol) / / HDL-cholesterol. CCC actually reflects the ratio of the content in the blood of atherogenic LP to antiatherogenic. This coefficient is no more than 1 in newborns, in healthy men of 20-30 years it reaches 2.5, in healthy women of the same age it is 2.2.In men 40-60 years old without clinical manifestations of atherosclerosis, Cx is between 3 and 3.5.In individuals with IHD, he is more than 4, often reaching 5-6.It is noteworthy that Cx is relatively low in long-livers: in individuals over the age of 90, it does not exceed 3. Cxc more accurately reflects the favorable and unfavorable combination of LP in terms of the risk of developing coronary artery disease and atherosclerosis.

When analyzing the results of the study, it should be borne in mind that an increase or decrease in the content of HDL-cholesterol is possible in a number of diseases or conditions).

However, the use of only HDL-XC values ​​to assess the risk of developing atherosclerosis can give potentially incorrect diagnostic information, so its values ​​should be assessed in comparison to the concentration of total cholesterol and LDL-C.

Low-density lipoprotein cholesterol in the blood serum

LD low-density lipoprotein( LDL-XC) cholesterol is the main transport form of cholesterol.

Recommended concentrations of LDL-C in serum for adults are 65-175 mg / dl or 1.68-4.53 mmol / l.

The study of LDL-cholesterol is performed for the purpose of phenotyping GLP, or dyslipoproteinemia( a modern term that replaces the old one - hyperlipoproteinemia).

LDL-cholesterol more closely correlates with the risk of developing atherosclerosis and ischemic heart disease than the concentration of total cholesterol. Low risk is noted at the concentration of LDL-C, less than 3.37 mmol / L, moderate - at 3.37-4.27 mmol / l, high - at a value exceeding 4.27 mmol / l. LDL-cholesterol can be determined by the calculation method according to the Friedvald formula: LDL-XC( mmol / L) = Total HD-HDL-XC-TG / 2.18.This method can not be used at a TG concentration above 4.52 mmol / l and in patients with type III HLP.

Atherogenicity of cholesterol is primarily determined by its belonging to a particular class of LP.In this regard, especially should be allocated to LDL, which are the most atherogenic for a number of reasons.

LDL transport two-thirds of the total plasma cholesterol and the richest in them( the content of cholesterol in them can reach up to 45-50%).The particle size( diameter 21-25 nm) allows LDL, along with HDL, to penetrate into the wall of the vessel through the endothelial barrier, but unlike HDL, which are easily removed from the wall, contributing to the removal of excess lipids, LDL are retained in it, because they have selective affinityto gluco-kozoaminoglycans and smooth muscle cells. The latter is explained by the presence of apo-B in the LDL, and on the surface of the cells the walls of the receptor vessel to the latter. For these reasons, LDL is the main transport form of cholesterol for the needs of vascular wall cells, and under pathological conditions, the source of its accumulation in the vessel wall. That is why in case of type II HLP, characterized by a high concentration of LDL-cholesterol, an early and severe atherosclerosis and coronary artery disease are often observed. The definition of LDL-cholesterol is very informative, and the deviation of this indicator from the norm can with a high degree of probability indicate what the danger with respect to the development of atherosclerosis and IHD.